Results 91 to 100 of about 181,836 (188)
Fatigue and Muscle Atrophy in a Mouse Model of Myasthenia Gravis Is Paralleled by Loss of Sarcolemmal nNOS [PDF]
, 2012 Myasthenia Gravis (MG) patients suffer from chronic fatigue of skeletal muscles, even after initiation of proper immunosuppressive medication. Since the localization of neuronal nitric oxide synthase (nNOS) at the muscle membrane is important for ...
Meinen, Sarina +7 more
core +2 more sources
The Effect of Corticosteroids on the Progression of Ocular Myasthenia to Generalized Myasthenia
, 1999 In previously published reports, 49% to 69% of patients with Ocular Myasthenia (OMG) will progress to Generalized Myasthenia Gravis (GMG without any medical intervention. Of the patients with only ocular manifestations at onset who later develop symptoms
Nicholas T. Monsul; Amy M. Knorr; Jonathan M. Goldstein; Robert L. Lesser
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Ocular Myasthenia Gravis and Thymus Gland Hyperplasia–A Case Report
TNOA Journal of Ophthalmic Science and ResearchMyasthenia gravis (MG) is caused by antibodies directed at acetylcholine receptors or functionally related proteins in the postsynaptic membrane at the neuromuscular junction.
Sujit Das
doaj +1 more source
Acta Ophthalmologica, Volume 104, Issue 6, Page e599-e614, September 2026.Abstract Purpose Immune checkpoint inhibitors (ICIs) have transformed cancer therapy but may cause immune‐related adverse events (irAEs), including dry eye disease (DED). This study aimed to quantify the incidence of ICI‐associated DED and to evaluate factors contributing to variability across studies.
Kai‐Yang Chen +2 more
wiley +1 more source
Ocular Myasthenia Gravis: A Case Report
Background: Myasthenia gravis (MG) is an autoimmune disease caused by faulty transmission at the neuromuscular junction leading to fatigable muscular weakness.
Kuiken, Carla Gilbertson, OD +1 more
core +1 more source
Clinical Variability and Genotype‐Driven Outcomes in CHRND‐Related Congenital Myasthenic Syndrome
European Journal of Neurology, Volume 33, Issue 9, September 2026.Clinical variability in CHRND‐related congenital myasthenic syndrome ranges from isolated ocular involvement to severe neonatal‐onset disease with respiratory insufficiency. In a multicenter cohort of nine patients, ocular symptoms represented the core phenotype, while disease severity was influenced by genotype and presumed residual acetylcholine ...
David Muhmann +16 more
wiley +1 more source
EFNS/ENS Guidelines for the treatment of ocular myasthenia.
, 2014 BACKGROUND AND PURPOSE: The symptoms of acquired autoimmune ocular myasthenia are restricted to the extrinsic eye muscles, causing double vision and drooping eyelids.
Evoli, Amelia
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European Journal of Neurology, Volume 33, Issue 9, September 2026.ABSTRACT Objectives TUBA4A pathogenic variants are associated with ALS, frontotemporal dementia, spastic ataxia, spasticity, ataxia, Parkinson's disease, female infertility, macrothrombocytopenia, and myopathy. Four recently reported patients with TUBA4A neonatal/childhood onset myopathy had also a decrement on repetitive nerve stimulation (RNS), but ...
Margherita Milone +7 more
wiley +1 more source
Intravenous Methylprednisolone Treatment of Ocular Myasthenia Gravis
, 2006 Ocular myasthenia gravis is a chronic disease generally requiring years of treatment. Treatment consists of either Mestinon or immunosuppressive agents. Many patients with ocular myasthenia gravis are elderly and long term prednisone treatment is fraught
K. Warden; I. Ben-Bassat Mizrachi; W. Cornblath
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Chlorpromazine induced ocular myasthenia gravis [PDF]
, 2017 Drug induced bilateral ptosis is a very rare adverse drug reaction. Here we report a case of ten year old male child with chlorpromazine induced bilateral ptosis due to ocular ...
S, Nitya +4 more
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