Hurdles to open access publishing faced by authors: a scoping literature review from 2004 to 2023. [PDF]
Kaliuzhna N +3 more
europepmc +1 more source
Publishing at any cost: a cross-sectional study of the amount that medical researchers spend on open access publishing each year. [PDF]
Ellingson MK +6 more
europepmc +1 more source
Finding novel vulnerabilities of hypomorphic BRCA1 alleles
Synthetic lethality screens performed to identify novel vulnerabilities often model complete gene loss, thereby overlooking patient‐derived hypomorphic mutations. In this study, we have performed genome‐wide CRISPR screens on BRCA1 hypomorphic mutations, showing BRCA1I26A behaves like wild‐type, while BRCA1R1699Q mimics deficiency. Furthermore, we have
Anne Schreuder +10 more
wiley +1 more source
Economic and geographical disparities in global contribution to open access publishing in anaesthesiology: A bibliometric analysis. [PDF]
Slawka E +6 more
europepmc +1 more source
Re: Jena R, Sharma AP, Madhavan K, Sridhar AN, Parmar K, Shrivastava N. What should urologists know about pseudojournals and open access publishing? A narrative review of the literature. Indian J Urol 2022;38:184-90. [PDF]
Kumar N, Tillu ND, Jain S.
europepmc +1 more source
Many patients with urothelial cancer do not benefit from treatment with pembrolizumab, while at risk of severe side effects. Changes in the levels of circulating tumor DNA early during treatment, measured by a simple and affordable assay that can be easily implemented in the clinic, can be used as a prognostic tool to identify these patients.
Youssra Salhi +14 more
wiley +1 more source
High-Impact Open Access Scientific Publishing
Manoel Antônio dos Santos
doaj +1 more source
Looking back at 20 years of Open Access publishing at Nucleic Acids Research. [PDF]
Roberts R.
europepmc +1 more source
Oncogenic DMTF1β promotes cancer cell motility by regulating autophagy through ULK1 stabilization
In the current study, we demonstrate that the oncogene DMTF1β regulates ULK1 stability by reducing its proteasomal degradation in cancer cells. This stabilization enables ULK1 to induce autophagy, which in turn facilitates cancer cell migration. Consequently, reduced DMTF1β levels lead to decreased autophagy and impaired cancer cell migration.
Jun Xu +13 more
wiley +1 more source

