Results 131 to 140 of about 7,602 (174)

Rapid detection of Mycoplasma hyopneumoniae by recombinase-aided amplification combined with the CRISPR/Cas12a system. [PDF]

Front Cell Infect Microbiol
Li K, Luo T, Zhang Y, Li C, Chen H, Xia C, Gao C.   +6 more
europepmc   +1 more source

Causal validation of the relationship between air pollution and lung cancer: A bidirectional Mendelian randomization study and meta-analysis. [PDF]

Medicine (Baltimore)
Wang X, Xiao G, Xu W, Ni C.
europepmc   +1 more source

Invisible Pain, Visible Inequalities: Gender, Social Agency, and the Health of Women with Fibromyalgia. [PDF]

Healthcare (Basel)
Lizama-Lefno A, Roco-Videla Á, Atenas-Núñez E, González-Droguett N, Muñoz-Yánez MJ, Flores-Carrasco SV.   +5 more
europepmc   +1 more source

Diversity and prevalence of dairy streptococcal phages in two European dairy fermentation facilities over a one-year period. [PDF]

Microb Genom
White K, Szafran JD, McDonnell B, Chawande G, Faherty-McGrath L, Lugli GA, Ventura M, Eraclio G, Volonté F, Dal Bello F, van Sinderen D, Mahony J.   +11 more
europepmc   +1 more source

Transcatheter Pulmonary Valve Implantation in Patients with Quadricuspid Pulmonary Valve Regurgitation. [PDF]

Anatol J Cardiol
Alsawidi H, Haidarah A, Mao C, Yuan F.
europepmc   +1 more source

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Modulation of p36 gene expression in human neuronal cells

Journal of the Neurological Sciences, 1995
p36 is a calcium/lipid-binding phosphoprotein that is expressed at high levels in proliferating and transformed cells, and at low levels in terminally differentiated cells, such as CNS neurons. The calcium-dependent binding to membrane phospholipids, and its capacity to interact with intermediate filament proteins suggest that p36 may be involved in ...
Alain Puisieux, S M De La Monte, J R Wands   +2 more
exaly   +3 more sources

P36 A CHALLENGING CASE OF MINOCA

European Heart Journal Supplements, 2023
Abstract Myocardial infarction in the absence of obstructive coronary artery disease (MINOCA) accounts for 5–6% of all myocardial infarctions. The causes that can lead to this diagnosis are many and not always clearly identifiable; in fact, despite optimal work–up, the cause of MINOCA remains undetermined in 8–25% of patients and ...
G Zagarese, A Candelora, G Vinco, C Giovannelli, C Angheben, P Moggio, G Musuraca, C Cemin, M Del Greco   +8 more
openaire   +1 more source

P36
A Novel RhD Variant

Transfusion Medicine, 2006
Introduction  Variants of the RhD antigen are often categorised into weak D and partial D phenotypes, although the distinction between the two groups is unclear. One frequently used definition is the ability to make alloanti‐D in partial D, but not in weak D phenotypes, however some phenotypes characterised as weak D are now known to be associated with
L. Tilley, T. Bullock, R. Mason, J. Poole, G. Daniels   +4 more
openaire   +1 more source

Phosphorylation of P36 in vitro by protein kinase C

Biochemical and Biophysical Research Communications, 1986
The 36kDa subunit of protein I (p36) is a major substrate of several tyrosine protein kinases. Here we demonstrate that protein kinase C catalyzes the incorporation of 1.7 moles of phosphate per mole of protein I. Phosphorylation is absolutely dependent on the presence of both calcium and phospholipid, and is specific for serine and threonine residues.
N C, Khanna, M, Tokuda, S M, Chong, D M, Waisman   +3 more
openaire   +2 more sources