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Correction to Supporting Information for Li et al., p53 deficiency induces MTHFD2 transcription to promote cell proliferation and restrain DNA damage. [PDF]
Proc Natl Acad Sci U S Aeuropepmc +1 more source
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Surgery for Obesity and Related Diseases, 2006
In this essay, the germline and somatic mutations data bases of tumor suppressor gene p53 were analyzed. The data bases can be found in the internet. The analysis was performed with the help of computer programms Microsoft Excel and iMars. The results were statistically analyzed, as well.
John B. Marshall +7 more
+7 more sources
In this essay, the germline and somatic mutations data bases of tumor suppressor gene p53 were analyzed. The data bases can be found in the internet. The analysis was performed with the help of computer programms Microsoft Excel and iMars. The results were statistically analyzed, as well.
John B. Marshall +7 more
+7 more sources
p53-Induced Apoptosis and Inhibitors of p53
Current Medicinal Chemistry, 2009Protein p53 is a key player in mitochondrial mediated apoptotic cell death and excess p53 activity has been implicated in many disease states such athrosclerosis, diabetes, osteoarthritis, Alzheimer's disease, Parkinson's disease, Huntington's disease, AIDS, P. falciparum and S. typhimurium infections.
Surendra Kumar, Nayak +2 more
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p53 transactivation through various p53-responsive elements
Molecular Carcinogenesis, 1996p53 is a nuclear phosphoprotein whose function is classified as tumor suppression. Studies have shown that p53 functions by binding to p53 DNA recognition sequences and regulates transcription of growth-regulatory genes. Various p53 recognition sequences have recently been identified.
D J, Park +4 more
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Gan to kagaku ryoho. Cancer & chemotherapy, 1997
The gene coding p53 is commonly affected by deletions, rearrangements, or point mutations in a variety of human cancers. p53 is a nuclear phosphoprotein. Mutations are frequently found at highly conserved residues of the p53 protein. The mutant p53 proteins examined so far each have a much longer half-life than that of the wild-type p53 protein which ...
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The gene coding p53 is commonly affected by deletions, rearrangements, or point mutations in a variety of human cancers. p53 is a nuclear phosphoprotein. Mutations are frequently found at highly conserved residues of the p53 protein. The mutant p53 proteins examined so far each have a much longer half-life than that of the wild-type p53 protein which ...
openaire +3 more sources
2019
Aim: this study was devoted to investigate the expression of some regulatory proteins of cell cycle in tumors with p53 high and negative expression, and comparison of these indices with both morphological characteristics of serous ovarian cancer (OC) and patients??? clinical characteristics.
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Aim: this study was devoted to investigate the expression of some regulatory proteins of cell cycle in tumors with p53 high and negative expression, and comparison of these indices with both morphological characteristics of serous ovarian cancer (OC) and patients??? clinical characteristics.
openaire +1 more source