Results 111 to 120 of about 11,309,322 (372)
Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs
In HGSOC with normal KRAS expression, high PTP4A3 expression regulates autophagy activation. Conversely, in HGSOC with high KRAS expression, KRAS dictates autophagy control, and PTP4A3 is not required. When high PTP4A3 expression is inhibited, HGSOC cells are preferentially sensitised towards DNA‐damaging agents.
Ana López‐Garza+3 more
wiley +1 more source
Kostikas K, Price D, Gutzwiller FS, et al. Int J Chron Obstruct Pulmon Dis. 2020;15:1729–1738. On page 1730 in Patients and Methods, Study Design section, and on page 1736 Ethics Approval section, the ISAC number reported was ...
Kostikas K+8 more
doaj
Miscellaneous front pages, Bull. Amer. Math. Soc., Volume 46, Number 2 (1940) [PDF]
openalex +1 more source
Every walk’s a hit: making page walks single-access cache hits
Chang Hyun Park+3 more
semanticscholar +1 more source
'Exhibition of Industry of All Nations in 1851' [PDF]
Page number(s) as given: pp.146 ...
core
Screening 166 FDA‐approved anticancer drugs identifies the aromatase inhibitor Exemestane as a synergistic partner of PARP inhibitor Olaparib in BRCA‐proficient triple‐negative breast cancer. Exemestane induces ROS‐mediated replication stress, enhancing DNA damage and apoptosis alongside Olaparib.
Nur Aininie Yusoh+5 more
wiley +1 more source
Miscellaneous front pages, Bull. Amer. Math. Soc., Volume 46, Number 5 (1940) [PDF]
openalex +1 more source
'A Looking-Glass for London - No. XXX London Extremes - Hyde Park and Rag Fair' [PDF]
Page number(s) as given: pp.497 ...
core
A‐to‐I editing of miRNAs, particularly miR‐200b‐3p, contributes to HGSOC progression by enhancing cancer cell proliferation, migration and 3D growth. The edited form is linked to poorer patient survival and the identification of novel molecular targets.
Magdalena Niemira+14 more
wiley +1 more source