Results 161 to 170 of about 87,991,808 (290)
A peer-2-peer electronic health record system streamlining healthcare delivery during short-term medical mission clinics. [PDF]
Lo SEJ, Chan LG.
europepmc +1 more source
PEER D5.3 USAGE STUDY Randomised controlled trial results
This report reviews the findings of an experiment to measure the effect of exposing early article versions in repositories on downloads of the version of record at various publishers' web sites. The research design used was a randomised controlled trial (
Rowlands, Ian +2 more
core
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi +6 more
wiley +1 more source
Decisions about risk taking: Elaborate dynamics between guests and hosts of peer-to-peer accommodation during COVID-19. [PDF]
Hu A, Pang J, Gan X.
europepmc +1 more source
Partial inhibition of focal adhesion kinase (FAK) can paradoxically promote tumor growth, rather than simply producing a weaker antitumor effect than that observed with strong FAK suppression. In breast cancer and melanoma models, targeting p110δ PI3K, particularly in macrophages, counteracted these tumor‐promoting effects, highlighting the importance ...
Lydia Xenou +4 more
wiley +1 more source
Negotiating privacy and responsibility in digital public health: a qualitative study of the social and ethical implications of peer-to-peer health data sharing. [PDF]
Lindberg J +3 more
europepmc +1 more source
Arginine methylation can be viewed as a persistence‐prone post‐translational modification regulated by a network of PRMTs. Competitive and compensatory interactions among PRMTs can redistribute methylation across substrate pools shaped by sequence, structural, spatial, and environmental layers, reinforcing RNA‐processing, chromatin, and signaling ...
So Hyun Kwon, Ji Min Lee
wiley +1 more source
Mechanisms and therapeutic opportunities of the ribotoxic stress response in cancer
Cancer cells' high translational demand creates opportunities to therapeutically target ribosome function. Ribosome stalling and collisions activate ZAKα and the ribotoxic stress response (RSR), which can trigger rapid, p53‐independent apoptosis in cancer.
Anastassiya Kim +7 more
wiley +1 more source
Medical Students as Medical Spanish Standardized Patients for Peer-to-Peer Communication Skills Practice and Feedback. [PDF]
Bresciani D +10 more
europepmc +1 more source

