Results 11 to 20 of about 16,214 (225)

Pemphigus vulgaris

open access: yesContemporary Clinical Dentistry, 2011
Pemphigus vulgaris is a chronic autoimmune mucocutaneous disease that initially manifests in the form of intraoral lesions, which spread to other mucous membranes and the skin. The etiology of pemphigus vulgaris is still unknown, although the disease has
Sandhya Tamgadge   +4 more
doaj   +3 more sources

Pemphigus Vulgaris [PDF]

open access: yesThe Journal of Emergency Medicine, 2019
Pemphigus vulgaris is a chronic autoimmune bullous dermatosis that results from the production of autoantibodies against desmogleins 1 and 3. It is the most frequent and most severe form of pemphigus, occurring universally, usually between 40 and 60 years of age.
Scarlet Charmelo, Silva   +3 more
  +11 more sources

Oral Pemphigus Vulgaris [PDF]

open access: yesCureus, 2021
Pemphigus is a chronic mucocutaneous autoimmune disease with the clinical feature of blisters that initially appear in the oral cavity and later in the skin. The dental professionals play an important role in diagnosing the disease. Early diagnosis and treatment determine the course and prognosis of the disease.
Subadra, K   +2 more
openaire   +2 more sources

Mucosal-dominant pemphigus vulgaris in a captopril-taking woman with angioedema [PDF]

open access: yesAnais Brasileiros de Dermatologia, 2015
We describe a 39-year-old woman with an apparent captopril-induced, contact mucosal-dominant pemphigus vulgaris and angioedema, who took captopril during a bout of arterial hypertension.
Justyna Gornowicz-Porowska   +3 more
doaj   +2 more sources

Relationship Between Pemphigus Vulgaris Severity and PCR-positive Herpes Simplex Virus

open access: yesActa Dermato-Venereologica, 2022
Pemphigus vulgaris is a rare autoimmune skin disease. Although herpes simplex virus has been associated with autoimmune diseases, evidence regarding its association with pemphigus vulgaris exacerbations is scarce. This retrospective cohort study aimed to
Sharon Baum   +7 more
doaj   +1 more source

Clinical periodontal diagnosis

open access: yesPeriodontology 2000, EarlyView., 2023
Abstract Periodontal diseases include pathological conditions elicited by the presence of bacterial biofilms leading to a host response. In the diagnostic process, clinical signs such as bleeding on probing, development of periodontal pockets and gingival recessions, furcation involvement and presence of radiographic bone loss should be assessed prior ...
Giovanni E. Salvi   +5 more
wiley   +1 more source

Pemphigus Vulgaris: A Clinical Study of 31 Cases (2004–2014) in Morocco

open access: yesDermatology Research and Practice, 2020
Background. Pemphigus vulgaris is a rare bullous autoimmune dermatosis whose evolution and prognosis are unpredictable. Aim. The objective was to analyze long-term outcomes in patients with pemphigus vulgaris by identifying the factors that are able to ...
Titou Hicham   +4 more
doaj   +1 more source

Pemphigus Vulgaris

open access: yes, 2022
Pemphigus is a group of chronic mucocutaneous blistering diseases caused by autoantibodies directed against the desmosomal cadherins desmoglein 1 and/or desmoglein 3 (Table 8.1). Pemphigus can be divided into two major forms, pemphigus foliaceus (PF) and pemphigus vulgaris (PV).
van der Wier, Gerda   +2 more
openaire   +2 more sources

Pemphigus vulgaris and focal segmental glomerulosclerosis (FSGS): First reported case and a review of the literature

open access: yesClinical Case Reports, 2023
Key Clinical Message There may be a connection between pemphigus vulgaris and nephrotic syndrome, as evidenced by the occurrence of focal segmental glomerulosclerosis in our pemphigus vulgaris patient and reviewing relevant literature.
Afsaneh Sadeghzadeh‐Bazargan   +5 more
doaj   +1 more source

Loss of flotillin expression results in weakened desmosomal adhesion and Pemphigus vulgaris-like localisation of desmoglein-3 in human keratinocytes [PDF]

open access: yes, 2016
Desmosomes are adhesion plaques that mediate cell-cell adhesion in many tissues, including the epidermis, and generate mechanical resistance to tissues. The extracellular domains of desmosomal cadherin proteins, desmogleins and desmocollins, are required
Ali, Jawahir   +7 more
core   +1 more source

Home - About - Disclaimer - Privacy