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Hyperkalemic Periodic Paralysis

Archives of Neurology, 1967
IN 1886 Eulenberg 1 described "paramyotonia congenita," an inherited syndrome in which episodes of myotonia and weakness were induced by exposure to cold. A similar condition was reported independently by Rich. 2 During the ensuing years additional cases were published, emphasizing the myotonia rather than the episodic weakness which was also part of ...
R B, Layzer, R E, Lovelace, L P, Rowland
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Hypokalemic Periodic Paralysis

Southern Medical Journal, 1984
Hypokalemic periodic paralysis is an unusual disease that may begin dramatically. Although terrifying to the patient, the attacks can usually be controlled if the proper diagnosis is made. Although much has been done to determine the pathogenesis, many questions remain unanswered.
V, Johnson, W W, Winternitz
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Thyrotoxic Periodic Paralysis

New England Journal of Medicine, 2021
Thyrotoxic Periodic Paralysis A 25-year-old man presented with sudden limb paralysis. Laboratory studies revealed a potassium level of 1.6 mmol per liter.
Michael Fralick, Shohinee Sarma
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Periodic paralysis

2018
The periodic paralyses are a group of skeletal muscle channelopathies characterizeed by intermittent attacks of muscle weakness often associated with altered serum potassium levels. The underlying genetic defects include mutations in genes encoding the skeletal muscle calcium channel Cav1.1, sodium channel Nav1.4, and potassium channels Kir2.1, Kir3.4,
Doreen, Fialho   +2 more
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Thyrotoxic periodic paralysis

The American Journal of Medicine, 1986
A case of thyrotoxic periodic paralysis is reported in a Hispanic man with an unusual recurrence six weeks after radioactive iodine treatment. Thyrotoxic periodic paralysis has now been well characterized in the literature: it occurs primarily in Orientals with an overwhelming male preponderance and a higher association of specific HLA antigens ...
J E, Ferreiro, D J, Arguelles, H, Rams
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Thyrotoxic periodic paralysis

The American Journal of Medicine, 1969
Abstract In an Oriental patient with thyrotoxic periodic paralysis, six weeks of therapy with reserpine alone returned the pulse rate to normal and reduced nervousness but did not prevent the attacks of periodic paralysis. These observations imply that hyperactivity of the sympathetic nervous system is not an essential feature of the mechanism by ...
J S, Resnick, J D, Dorman, W K, Engel
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