Results 151 to 160 of about 241,755 (405)
Multi‐ciliated cells in the infundibulum and ampulla (INF/AMP) epithelium are vacuolated in aging. Unique cellular susceptibility of the INF/AMP epithelial population and aging‐associated decline in ovarian artery circulation, which supports the ovary and INF/AMP, contribute to this region‐specific vacuolation phenotype, as a consequence of a mildly ...
Keerthana Harwalkar+10 more
wiley +1 more source
Analytical Studies on the TSH-releasing Activity of Posterior Pituitary Hormone
Naohisa Hiroshige
openalex +2 more sources
Effects of theophylline and adenosine 3′:5′-cclic monophosphate on growth hormone release from the isolated rat anterior pituitary gland [PDF]
Robin Ewart, K. W. Taylor
openalex +1 more source
Modulation of Pituitary Luteinizing Hormone Releasing Hormone Receptors by Sex Steroids and Luteinizing Hormone Releasing Hormone in the Rat [PDF]
MARCHETTI, Bianca Maria+3 more
openaire +4 more sources
Autophagy regulates ovarian reserve, with Cathepsin B playing a critical role in aging. Inhibition of Cathepsin B increases follicle count, enhances IGF1R–AKT–mTOR signaling and improves mitochondrial function without compromising fertility. This conserved mechanism suggests potential therapeutic strategies to delay ovarian aging and sustain fertility,
Aradhana Mohanty+8 more
wiley +1 more source
Circadian Gene BMAL1 Regulation of Cellular Senescence in Thyroid Aging
BMAL1 downregulation during aging reduces NFKBIA expression, accelerating thyroid cellular senescence and impairing hormone synthesis. Circadian rhythm disruption exacerbates thyroid functional decline, highlighting the BMAL1‐NFKBIA axis as a key mediator of aging‐related thyroid dysfunction and offering insights for stabilizing thyroid health in aging
Dandan Zong+4 more
wiley +1 more source
Receptor-mediated internalization of fluorescent gonadotropin-releasing hormone by pituitary gonadotropes. [PDF]
Eli Hazum+3 more
openalex +1 more source
A Novel GH Deficient Rat Model Reveals Cross‐Species Insights Into Aging
Mouse studies show disrupted GH signaling reduces body size, increases longevity, and improves health. We developed a CRISPR/Cas9 GH‐deficient rat model to address inconsistent rat evidence. GH‐deficient rats mirrored these findings, showing reduced body weight, increased body fat, enhanced insulin sensitivity, and altered gut microbiomes.
Soe Maung Maung Phone Myint+5 more
wiley +1 more source