Results 161 to 170 of about 2,251,238 (297)

Commodity risk assessment of <i>Lonicera ligustrina</i> var. <i>pileata</i>, <i>Lonicera ligustrina</i> var. <i>yunnanensis</i> and <i>Lonicera periclymenum</i> plants from the UK. [PDF]

open access: yesEFSA J
EFSA Panel on Plant Health (PLH)   +33 more
europepmc   +1 more source

In vitro properties of patient serum predict clinical outcome after high dose rate brachytherapy of hepatocellular carcinoma

open access: yesMolecular Oncology, EarlyView.
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser   +14 more
wiley   +1 more source

Arboviruses manipulate rice's volatile emissions, protecting insect vectors from natural enemies in the field. [PDF]

open access: yesSci Adv
Liu Q   +9 more
europepmc   +1 more source

Antioxidative and Free Radical Scavenging Activities of Some Plants Used in Thai Folk Medicine [PDF]

open access: bronze, 2006
Nongporn Hutadilok‐Towatana   +4 more
openalex   +1 more source

A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression

open access: yesMolecular Oncology, EarlyView.
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak   +8 more
wiley   +1 more source

Adaptaquin is selectively toxic to glioma stem cells through disruption of iron and cholesterol metabolism

open access: yesMolecular Oncology, EarlyView.
Adaptaquin selectively kills glioma stem cells while sparing differentiated brain cells. Transcriptomic and proteomic analyses show Adaptaquin disrupts iron and cholesterol homeostasis, with iron chelation amplifying cytotoxicity via cholesterol depletion, mitochondrial dysfunction, and elevated reactive oxygen species.
Adrien M. Vaquié   +16 more
wiley   +1 more source

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