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Elevated Plasmin(ogen) as a Common Risk Factor for COVID-19 Susceptibility
Physiological Reviews, 2020 Patients with hypertension, diabetes, coronary heart disease, cerebrovascular illness, COPD, and kidney dysfunction have worse clinical outcomes when infected with SARS-CoV-2, for unknown reasons.
Hong-Long Ji +2 more
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Physiological Reviews, 2020 To the Editor: Ji et al. (6) have convincingly shown that elevated plasmin(ogen) is a common risk factor in COVID-19 susceptibility. They highlighted the clinical relevance of plasmin(ogen) in patients who are susceptible to SARS-CoV-2 because of ...
Alain R Thierry
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Recent Advances on Plasmin Inhibitors for the Treatment of Fibrinolysis‐Related Disorders
Medicinal Research Reviews, 2014 Rami A Al-Horani, Umesh R Desai
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Neuronal Death in the Hippocampus Is Promoted by Plasmin-Catalyzed Degradation of Laminin
Cell, 1997 Excess excitatory amino acids can provoke neuronal death in the hippocampus, and the extracellular proteases tissue plasminogen activator (tPA) and plasmin (ogen) have been implicated in this death.
Sidney Strickland
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Plasmin-α2-plasmin inhibitor complex and α2-plasmin inhibitor in chronic subdural hematoma
Journal of Neurosurgery, 1989✓ Levels of the plasmin-α2-plasmin inhibitor complex (PLN-A2PI complex) and α2-plasmin inhibitor (A2PI) were determined by enzyme-linked immunosorbent assay (ELISA) with monoclonal antibodies in 59 patients with 66 chronic subdural hematomas (SDH's). Normal concentrations of the PLN-A2PI complex and A2PI in plasma are below 0.8 µg/ml and 60.5 ± 16.1 µg/
K, Saito +3 more
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Mechanism of plasmin generation by S100A10
Thrombosis and Haemostasis, 2017Summary Plasminogen (Pg) is cleaved to form plasmin by the action of specific plasminogen activators such as the tissue plasminogen activator (tPA). Although the interaction of tPA and Pg with the surface of the fibrin clot has been well characterised ...
Victoria A. Miller +8 more
semanticscholar +1 more source
Plasmin-α2-antiplasmin complexes (plasmin—plasmin inhibitor complexes)
1999α2-Antiplasmin (new name: plasmin inhibitor) is the most important plasmin inhibitor. Its rapid reaction with plasmin results in the formation of an inactive complex composed of one molecule of each component. Two steps are involved in this process: first a reversible complex is formed between the lysine-binding sites of plasmin and complementary sites
E. Hattey +4 more
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Plasmin inactivation in Plasma
Thrombosis and Haemostasis, 1976SummaryInhibition and inactivation of plasmin is ascribed to α2-macroglobulin, α1-antitrypsin and c1-esterase inhibitor. In an “overall” inactivation test of plasmin in plasma, which comes perhaps closest to rapid inactivation of plasmin in the circulating blood, we only found a correlation between the immunological concentration of α2-macroglobulin ...
E M, Haselager, T M, Goote, J, Vreeken
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Thrombosis Research, 1993
In order to understand the mechanism for the complex between alpha 2-plasmin-inhibitor (alpha 2-PI) and plasmin to express its specific activity on fibrin autography after SDS-PAGE, we analyzed the effects of SDS on alpha 2-PI molecule and alpha 2-PI-plasmin complex.
D, Yan, T, Urano, Y, Takada, A, Takada
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In order to understand the mechanism for the complex between alpha 2-plasmin-inhibitor (alpha 2-PI) and plasmin to express its specific activity on fibrin autography after SDS-PAGE, we analyzed the effects of SDS on alpha 2-PI molecule and alpha 2-PI-plasmin complex.
D, Yan, T, Urano, Y, Takada, A, Takada
openaire +2 more sources