Results 211 to 220 of about 32,923 (246)

Chloroquine Resistance in Plasmodium Berghei

The American Journal of Tropical Medicine and Hygiene, 1966
Summary A strain of Plasmodium berghei was passed through mice which were maintained on a diet containing 0.008% chloroquine diphosphate and which had been treated intravenously with ethyl palmitate to destroy or diminish the phagocytic functions of the spleen.
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Pathophysiology of Plasmodium berghei infection in mice

Experimental Parasitology, 1965
Abstract Significant biochemical changes were detected in the serum of mice infected with P. berghei . Marked increases in SGP-T and SGO-T transaminases were observed as early as 2 days after infection. Lower fasting glucose levels occurred in heavily infected mice 4 days after infection.
Elvio H. Sadun, J. S. Williams, Gerlinde Hutt, Frances C. Meroney   +3 more
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Interferon Induced by Plasmodium berghei

Science, 1968
An inhibitor of virus is demonstrable in the serums of mice infected with Plasmodium berghei . The titer of the inhibitor, detectable within 10 hours after injection of parasitized blood, increases rapidly until 30 to 40 hours when it levels off or decreases slightly before reaching a plateau.
Kun-Yen Huang, Francis B. Gordon, Warren W. Schultz   +2 more
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Plasmodium berghei: Energy metabolism of sporozoites

Experimental Parasitology, 1978
Abstract The energy metabolism of Plasmodium berghei sporozoites was studied by using their motility as an indicator of energy production and consumption. Sporozoites suspended in medium without sugars or amino acids ceased to move. Motility was restored by the addition of any of several sugars or amino acids to the medium. Inhibition of sporozoite
Jerome P. Vanderberg, Stephen R. Mack
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Plasmodium berghei Infections in Thymectomized Rats

Nature, 1968
THE course of P. berghei infections in the rat has been described many times1–4. Young rats, up to about 8 weeks old, usually die with a fulminating acute parasitaemia and severe anaemia. On the other hand, most adult rats develop immunity and almost completely clear the blood stream of parasites soon after the peak parasitaemia.
A. C. Allison, I. N. Brown, R. B. Taylor
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Exotoxin in malaria infection (Plasmodium berghei)

Experientia, 1967
Swiss Mause, die unbehandelt eine Infektion mitPl. berghei niemals uberleben, wurden mit Serum von frisch infizierten Tieren (12. Infektionstag) immunisiert und anschliessend infiziert. Obgleich sich danach in allen Fallen eine schwere Parasitamie entwickelte, heilten 44% derjenigen Tiere nach dem 35.
Chr. Jerusalem, D. Bruchhausen
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Enzyme variation in Plasmodium berghei

Transactions of the Royal Society of Tropical Medicine and Hygiene, 1970
Abstract 10 strains of P. berghei representing 4 subspecies—P.b. berghei, P.b. yoelii, P.b. killicki and a “P. berghei-like” parasite from Nigeria were used in this study. The electrophoretic band patterns of 4 enzymes—glucose phosphate isomerase, adenylate kinase, 6-phosphogluconate dehydrogenase and malate dehydrogenase were examined in all strains.
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Plasmodium berghei: Oxidant defense system

Experimental Parasitology, 1985
Glutathione oxidant defense system protects the erythrocyte from oxidative damage. This defense system was studied in mouse erythrocytes infected with Plasmodium berghei and in isolated parasites. The efficiency of this system was found to be increased in parasitized erythrocytes compared to the normal erythrocytes.
A.S. Saini, T.S. Jaswal, R.K. Seth
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Characterization of Plasmodium berghei glutathione synthetase

Parasitology International, 2011
Plasmodium berghei contained 0.454±0.031 U/mg of glutathione synthetyase (GS). GS was purified using solid ammonium sulfate and Sephadex G-200 from P. berghei infected mouse erythrocytes. SDS-PAGE showed purified GS as a single band protein of 70 kDa and its Km for γ-glutamylcysteine, glycine and ATP being 0.33 mM, 8.3 mM and 0.43 mM respectively with ...
H. S. Banyal, S.K. Sharma
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Plasmodium berghei: The in Vitro immune response

Experimental Parasitology, 1981
Abstract Following primary in vitro Stimulation by Plasmodium berghei, IgM titers by the indirect fluorescent antibody test (IFAT) were negative in in vitro reconstituted syngeneic mouse spleen cultures containing T cells and macrophages, or B cells and macrophages, or macrophages alone, but IgM titers of 1:20 were obtained from cultures containing B
Shong Wan Norby, Nelda E. Alger
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