Results 161 to 170 of about 29,127 (200)
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Complications of Acute Posterior Vitreous Detachment

Archives of Ophthalmology, 1968
An acute onset of suddenly appearing entopsias with or without light flashes usually is caused by a posterior vitreous detachment with collapse. Ordinarily this is an innocuous process. However, a study of 84 such patients revealed that three types of complications may be encountered: retinal breaks, intravitreal or retrovitreal hemorrhages or both ...
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Posterior vitreous detachment after neodymium:YAG laser posterior capsulotomy

Journal of Cataract and Refractive Surgery, 2003
To determine whether retinal detachment (RD) after neodymium:YAG (Nd:YAG) laser posterior capsulotomy is due to a greater incidence of posterior vitreous detachment (PVD) than in controls and whether vitreous status at the time of capsulotomy is useful in predicting the risk for RD.Teaching hospital ophthalmology service.Patients having Nd:YAG laser ...
Richard M, Sheard   +4 more
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Posterior Vitreous Detachment: A Combined Clinical and Physicochemical Study

Retina, 1985
Sixty-one postmortem eyes were examined with clinical methods (slit lamp) and later sectioned for measurement of the liquid and gel vitreous. The amount of liquid vitreous was found to be progressively greater with increasing posterior vitreous detachment (PVD), and it was concluded that slit-lamp examination is a reliable method of evaluating the ...
L, Larsson, S, Osterlin
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Complications of Acute Posterior Vitreous Detachment

American Journal of Ophthalmology, 1975
Of 201 patients whose presenting symptoms were acute entoptic phenomena or photopsia, or both, 150 patients had posterior vitreous detachment; 69 patients (46%) had retinal breaks; 18 (12%) had a vitreous hemorrhage without detectable retinal breaks; and two (1.3%) had peripheral retinal hemorrhages without retinal breaks or vitreous hemorrhage ...
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[Posterior vitreous detachment].

Klinische Monatsblatter fur Augenheilkunde, 1989
Posterior vitreous detachments (PVDs) are common. Their complications--retinal detachment--however, are comparatively rare. PVDs without complications and those prone to complications can be differentiated biomicroscopically by morphological signs, that is by examining the posterior hyaloid and its insertion at the posterior border of the vitreous base.
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Posterior vitreous detachment.

Optometry clinics : the official publication of the Prentice Society, 1993
Posterior vitreous detachment is an expected consequence of aging, but it can also be the initiating cause of a retinal detachment. To understand the mechanism of posterior vitreous detachment and its sequelae, it is necessary to appreciate the anatomy of the vitreous, its development, and the pathogenesis of vitreous degeneration.
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Ultrastructural features of posterior vitreous detachment

Albrecht von Graefes Archiv f�r Klinische und Experimentelle Ophthalmologie, 1975
The ultrastructural features of vitreoretinal relations in the posterior fundus of enucleated eyes were studied. Vitreous detachment occurs at the anatomical vitreoretinal juncture, between the inner limiting lamina of the retina and the vitreous cortex, the surface of the lamina becoming smooth and without vitreous residues.
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Vitreous shrinkage and posterior vitreous detachment

2011
Vitreous undergoes a progressive separation into a gel-like phase and a liquid phase with aging. This liquefaction process is very slow and results from a structural change in the network of colla- gen fibrils which are the main constituent of the vitreous gel [2]. The fibrils coalesce into bundles and let the liquid vitreous free to separate from them.
TATONE, Amabile, Testa A.
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II.C. Vitreous Aging and Posterior Vitreous Detachment

2014
Vitreous undergoes dramatic changes with age, the most notable called “synchysis senilis,” which refers to the liquefaction of the gel with age. An understanding of synchysis from molecular to macroscopic levels is crucial because it is directly responsible for many important pathologic conditions that form a large and diverse group of ocular diseases.
Kevin Tozer, Mark W. Johnson, J. Sebag
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