Results 141 to 150 of about 6,492 (168)

High-Intensity Interval Training Reduces Adipocyte Size and enhances the Thermogenic Activity of White and Brown Adipose Tissue in Male Wistar Rats. [PDF]

Int J Prev Med
Moein AR, Pourvaghar MJ, Kazeminasab F.
europepmc   +1 more source

Ontogeny and transcriptional regulation of Thetis cells. [PDF]

Nature
Paucar Iza YA, Park T, Baker E, Shibu G, Hoelting T, Feather G, Yadav A, Parisotto YF, Zhao Z, Akagbosu B, Bayes ME, Fisher L, James LM, Ma J, Philpot BD, Afzali B, Leslie C, Brown CC.   +17 more
europepmc   +1 more source

PCB-126 exposure promotes brown adipose tissue dysfunction and metabolic inflexibility in mice. [PDF]

Sci Rep
Peixoto TC, Miranda CS, Teixeira AMR, Quitete FT, Teixeira AVS, Monteiro EB, Martins BC, de Castro Resende A, Martins FF, de Barros Mucci D, Daleprane JB.   +10 more
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Alpha-ketoglutarate supplementation improves hyperglycemia and attenuates the decrease in GLUT4 and PGC-1α proteins in adipose tissue of streptozotocin-high-fat diet-induced diabetic mice. [PDF]

J Nutr Sci
Takemura A, Matsunaga Y, Hajime S, Wang W, Takahashi Y, Hatta H.   +5 more
europepmc   +1 more source

Functions of Prdm16 in thermogenic fat cells [PDF]

Temperature, 2015
The PR-domain containing 16 (Prdm16) protein is a powerful inducer of the thermogenic phenotype in fat cells. In both developmental (brown) and induced (beige) thermogenic adipose tissue, Prdm16 has a critical role in maintaining proper tissue structure and function.
Patrick Seale, Jeff Ishibashi
exaly   +3 more sources

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The Multifaceted Roles of PRDM16: Adipose Biology and Beyond

Trends in Endocrinology and Metabolism, 2016
The PRDM [PRDI-BFI (positive regulatory domain I-binding factor 1) and RIZ1 (retinoblastoma protein-interacting zinc finger gene 1) homologous domain containing] protein family is involved in a spectrum of biological processes including cell fate determination and development.
Paul Cohen, Jingyi Chi
exaly   +3 more sources

Role of Mel1/Prdm16 in bone differentiation and morphology

Experimental Cell Research, 2022
MEL1 (MDS1/EVI1-like gene 1/PRDM16), a zinc finger protein, is located near the chromosomal breakpoint at 1p36 in human acute myeloid leukemia (AML) cells with the t (1; 3) (p36; q21) translocation. Mel1/Prdm16 is not only a causative gene of leukemia, but also has multiple regulatory functions, such as the regulation of fat metabolism.
Kazuko Kaneda-Nakashima, Kaori Igawa, Mathurot Suwanruengsri, Fuke Naoyuki, Tomonaga Ichikawa, Taro Funamoto, Shuji Kurogi, Tomohisa Sekimoto, Yoshihiro Yamashita, Etsuo Chosa, Ryoji Yamaguchi, Kazuhiro Morishita   +11 more
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PRDM16: the interconvertible adipo-myocyte switch

Trends in Cell Biology, 2009
Both brown and white adipocytes were previously considered to be derived from the same precursor cell, despite being histologically and functionally different. However, a recent study shows that overexpression of the transcriptional regulator positive regulatory domain containing 16 (PRDM16) determines the development of brown adipocytes from a ...
Sesma, M.P. (María Pilar), Frühbeck, G. (Gema), Burrell, M.A. (María Ángela)   +2 more
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Prdm3 and Prdm16 cooperatively maintain hematopoiesis and clonogenic potential

Experimental Hematology, 2020
Mds1-Evi1 (also known as Prdm3) and Prdm16 are two highly related zinc finger transcription factors that, within the hematopoietic system, are both expressed primarily in hematopoietic stem cells (HSCs). Our laboratory previously found that constitutive Mds1-Evi1 knockout mice are viable, but their HSCs are unable to withstand myeloablative ...
Kelly A. McGlynn, Rongli Sun, Alin Vonica, Sarah Rudzinskas, Yi Zhang, Archibald S. Perkins   +5 more
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Dilated cardiomyopathy in patients with PRDM16 haploinsufficiency

Journal of Molecular Medicine
PRDM16 has been identified as a potential causal gene for cardiomyopathies, supported by reports of several cases associated with loss-of-function (LoF) variants in PRDM16. In this multi-centric study, we present the largest cohort to date of dilated cardiomyopathy (DCM) patients harboring PRDM16 LoF variants, including eleven previously unreported ...
Clarisse Billon, Gilles Millat, Adeline Goudal, Valerie Malan, Diala Khraiche, Karim Wahbi, Nadine Ferrier, Jean-Christophe Eicher, Romain Tixier, Nadir Benbrik, Océane Bouchot, Léa Gaudillat, Annabelle Venisse, Pascaline Berthome, Xavier Jeunemaitre, Damien Bonnet   +15 more
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