Results 261 to 270 of about 156,635 (305)
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Pregnancy-induced hypertension

1986
The best approach to the management of pregnancy-induced hypertension (PIH) would be its prevention (Table 17.1). Unfortunately, this goal will continue to elude us until we understand the primary pathogenesis of this condition. Some hypotheses are beginning to emerge.
F. Broughton Pipkin, E. M. Symonds
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Anesthesia for Pregnancy-Induced Hypertension

Clinics in Perinatology, 1982
Women with severe pregnancy-induced hypertension are often critically ill; their fetuses are usually compromised. An ideal anesthetic method does not exist for the parturient with severe hypertension, hypovolemia, and organ failure. Optimal anesthetic results depend upon thorough preanesthetic evaluation and best medical control of pathophysiology ...
A S, Wheeler, B A, Harris
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Labetalol pharmacokinetics in pregnancy-induced hypertension

American Journal of Obstetrics and Gynecology, 1990
Pharmacokinetic parameters of oral labetalol were studied in eight women with pregnancy-induced hypertension in the third trimester of pregnancy. Labetalol exhibited rapid absorption; peak serum concentrations of 881 +/- 219 ng/ml occurred at 20 minutes after labetalol ingestion.
R C, Rogers, B M, Sibai, W D, Whybrew
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Neutrophil activation in pregnancy‐induced hypertension

BJOG: An International Journal of Obstetrics & Gynaecology, 1989
Summary. Human neutrophil elastase may be a major mediator of vascular damage and could contribute to the vascular damage seen in women with pregnancy‐induced hypertension (PTH). Elevated plasma levels of this substance will reflect neutrophil activation in vivo.
I A, Greer   +4 more
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PLASMA PROLACTIN IN PREGNANCY INDUCED HYPERTENSION

BJOG: An International Journal of Obstetrics & Gynaecology, 1978
SummaryIn a prospective study, plasma prolactin concentration at 37 to 41 weeks gestation in 45 primigravidae showed a significant correlation with maximal rise in diastolic blood pressure between levels at 7 to 16 weeks and those measured between 28 weeks and delivery.
D M, Jenkins, L A, Perry
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Pregnancy‐induced hypertension: recurrence rate in second pregnancies

Medical Journal of Australia, 1991
The purpose of this study was to estimate the rate of recurrence of pregnancy-induced hypertension in an Australian population.Case records of women with hypertension during pregnancy who were delivered at our hospital during a two-year period were examined retrospectively.
J L, Hargood, M A, Brown
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Chronic Hypertension, Pregnancy-Induced Hypertension, and Low Birthweight

Epidemiology, 1994
We examined the associations between chronic hypertension, pregnancy-induced hypertension, and low birthweight in a retrospective cohort study using Washington birth certificate data. The adjusted prevalence ratio for low birthweight associated with chronic hypertension was 3.9 [95% confidence interval (CI) = 3.4-4.4].
P, Velentgas, E, Benga-De, M A, Williams
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Endothelial dysfunction after pregnancy‐induced hypertension

International Journal of Gynecology & Obstetrics, 2013
AbstractObjectiveTo carry out long‐term analysis of the presence of endothelial dysfunction after the development of pregnancy‐induced hypertension (PIH).MethodsIn a retrospective cohort study, data were analyzed from 60 women who delivered at a tertiary maternity hospital in Fortaleza, Ceara, Brazil, between 1992 and 2002.
Ana C P T, Henriques   +5 more
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Transient Blindness in Pregnancy Induced Hypertension

Asia-Oceania Journal of Obstetrics and Gynaecology, 1994
AbstractSudden blindness in pregnancy is a devastating experience for the patient, her family and the treating obstetrician. Four cases of cortical blindness with pregnancy induced hypertension are presented, seen at the General Hospital Kota Bharu, Kelantan, in 1990.
S, Achanna, D, Monga, , Sivagnanam
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Transient blindness in pregnancy induced hypertension

International Journal of Gynecology & Obstetrics, 1989
AbstractTransient blindness associated with pregnancy induced hypertension without neurological symptoms is a rare phenomenon. The blindness in these cases is postulated to be of “cortical blindness”. Two such cases occurring immediately after childbirth are presented. The ophthalmic manifestations of this complication of pregnancy induced hypertension
S, Nalliah, A S, Thavarashah
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