Results 221 to 230 of about 81,568 (349)
ABSTRACT In utero interventions are transformative in addressing genetic and anatomic conditions during fetal development. Next generation sequencing enables early genetic testing, playing a pivotal role in prenatal decision‐making by supporting risk stratification, precise and timely diagnosis, which directly informs eligibility for fetal surgical and
Matthew A. Shear +7 more
wiley +1 more source
Association of exposure to PM2.5-bound metals with premature rupture of membranes: a prospective cohort study. [PDF]
Liang M +15 more
europepmc +1 more source
Inflammatory predictors of neurologic disability after preterm premature rupture of membranes.
Jennifer Armstrong‐Wells +5 more
semanticscholar +1 more source
ABSTRACT Hemolytic disease of the fetus and newborn (HDFN) remains a significant concern in prenatal care primarily caused by maternal alloimmunization against fetal red blood cell antigens, most commonly the D antigen. Noninvasive fetal RHD genotyping, used as a screening tool, enables targeted antenatal prophylaxis and has been implemented in several
Emilie Thorup +4 more
wiley +1 more source
Low-dose oral misoprostol vs. oxytocin in premature rupture of membranes at ≥ 36 weeks: a comparison of labor induction outcomes and postpartum hemorrhage risk. [PDF]
Yang Y, Yu Z, Shi H, Wang Y, Zhao Y.
europepmc +1 more source
Evolving Features of RASopathies Among Pregnancies With Abnormal Fetal Fluid Collections
ABSTRACT Objective We aimed to characterize the fetal features across gestation and describe genotype‐phenotype correlations for pregnancies with fetal RASopathies that were more severely affected as they presented with at least one abnormal fluid collection.
Natalie B. Gulrajani +17 more
wiley +1 more source
Do we need to reconsider our antibiotic regimen for preterm premature rupture of membranes? Pathogens and perinatal outcome in PPROM. [PDF]
Schuler L +6 more
europepmc +1 more source
The Medico-Moral Problem Regarding Hydramnios* [PDF]
Schmitt, Alexander H.
core +1 more source

