Results 11 to 20 of about 2,330 (251)

A companion to the preclinical common data elements for proteomics, lipidomics, and metabolomics data in rodent epilepsy models. A report of the TASK3‐WG4 omics working group of the ILAE/AES joint translational TASK force

open access: yesEpilepsia Open, EarlyView., 2022
Abstract The International League Against Epilepsy/American Epilepsy Society (ILAE/AES) Joint Translational Task Force established the TASK3 working groups to create common data elements (CDEs) for various preclinical epilepsy research disciplines.
Laura Bindila   +11 more
wiley   +1 more source

Impacts of the combined exposure to seawater acidification and arsenic on the proteome of Crassostrea angulata and Crassostrea gigas [PDF]

open access: yes, 2018
Proteomic analysis was performed to compare the effects of Arsenic (As), seawater acidification (Low pH) and the combination of both stressors (Low pH + As) on Crassostrea angulata and Crassostrea gigas juveniles in the context of global environmental ...
Bebianno, Maria   +6 more
core   +1 more source

Linking physical activity to breast cancer:text mining results and a protocol for systematically reviewing three potential mechanistic pathways [PDF]

open access: yes, 2021
Epidemiological research suggests that physical activity is associated with a reduced risk of breast cancer, but the causal nature of this link is not clear.
Brown, Kristy A   +10 more
core   +1 more source

Male reproductive health and environmental xenoestrogens [PDF]

open access: yes, 1996
EHP is a publication of the U.S. government. Publication of EHP lies in the public domain and is therefore without copyright. Research articles from EHP may be used freely; however, articles from the News section of EHP may contain photographs or ...
A Giwercman   +89 more
core   +2 more sources

The Link between Environmental Toxicant Exposure and Endometriosis Re-Examined [PDF]

open access: yes, 2020
Endometriosis is widely acknowledged to be an estrogen dependent disease or unknown etiology. Recognition that environmental toxicants can bind with and activate the estrogen receptor, dysregulate steroid metabolism and, in some cases, act as anti ...
Foster, Warren G., Freger, Shay M.
core   +2 more sources

The E-SCREEN assay as a tool to identify estrogens: an update on estrogenic environmental pollutants. [PDF]

open access: yes, 1995
Estrogens are defined by their ability to induce the proliferation of cells of the female genital tract. The wide chemical diversity of estrogenic compounds precludes an accurate prediction of estrogenic activity on the basis of chemical structure ...
Chung, K L   +5 more
core   +4 more sources

Body Burdens of Polychlorinated Dibenzo-p-dioxins, Dibenzofurans, and Biphenyls and Their Relations to Estrogen Metabolism in Pregnant Women [PDF]

open access: yes, 2006
Polychlorinated dibenzo-p-dioxins (PCDDs, dioxins), polychlorinated dibenzofurans (PCDFs), and polychlorinated biphenyls (PCBs) are environmental endocrine disruptors that have half-lives of 7–10 years in the human body and have toxicities that probably ...
Chien-Ming Li   +8 more
core   +2 more sources

Effect of PCBs in plasma on risk of postmenopausal breast cancer [PDF]

open access: yes, 2021
Background: Polychlorinated biphenyls (PCBs) are a group of persistent organic pollutants suspected to cause negative health effects such as cancer. However, the findings on breast cancer are inconsistent, and lack data on larger populations.
Opsal, Tonje Koldal
core  

Adipose Concentrations of Organochlorine Compounds and Breast Cancer Recurrence in Long Island, New York [PDF]

open access: yes, 2003
Several studies have measured the association between blood or adipose concentrations of organochlorinated compounds (OCs), such as pesticides and polychlorinated biphenyls (PCBs), and breast cancer.
Britton, Julie A.   +7 more
core   +2 more sources

Polynuclear aromatic hydrocarbon carcinogens as antiestrogens in MCF-7 human breast cancer cells: role of the Ah receptor [PDF]

open access: yes, 1992
Treatment of MCF-7 cells with 1.0 μM 3-methylcholanthrene (MC) caused a decrease in cell proliferation and [3H]thymidine uptake whereas no effects were observed at a lower (0.1 μM) concentration.
Chaloupka, K., Krishnan, V., Safe, S.
core   +1 more source

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