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Research progress on programmed cell death of cardiomyocytes in pressure-overload hypertrophic cardiomyopathy. [PDF]

open access: yesApoptosis
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Programmed Cell Death

2006
Apoptosis is a physiological process of cell suicide that is implemented by activation of latent proteases that demolish vital cellular constituents. Apoptosis is used to sculpt the body during development, to maintain constant cell number by balancing cell production by mitosis, and to remove cells that are damaged or infected.
David L. Vaux, Andreas Strasser
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PROGRAMMED CELL DEATH

Transplantation, 1998
Programmed cell death (PCD) is currently one of the most intensively studied areas in cell biology. Substantial evidence now exists demonstrating the integral role of PCD in many fundamental immunologic processes; therefore, understanding the mechanisms of PCD may provide advances with broad implications in immunobiology.
E S, Woodle, S, Kulkarni
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Programmed Cell Death

Science, 1996
Letters from: [ Jan Novak ][1] [ Alice B. Fulton ][1] [ Jean Claude Ameisen ][1] Jean Claude Ameisen's Perspective “The origin of programmed cell death [PCD]” ([31 May, p. 1278][2]) discusses possible evolutionary roots of cell suicide.
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Erythrocyte programmed cell death

IUBMB Life, 2008
AbstractEryptosis, the suicidal death of erythrocytes, is characterised by cell shrinkage, membrane blebbing and cell membrane phospholipid scrambling with phosphatidylserine exposure at the cell surface. Phosphatidylserine‐exposing erythrocytes are recognised by macrophages, which engulf and degrade the affected cells.
Michael, Föller   +2 more
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Programmed Cell Death

Deutsche medizinische Wochenschrift (1946), 1997
How Hormones Regulate Programmed Cell Death During Amphibian Metamorphosis J.R. Tata. Activation of Matrix Metalloproteinase Genes during Thyroid Hormne-Induced Apoptotic Tissue Remodeling Y.-B. Shi, A. Ishizuya-Oka. Mechanisms by Which Matric Metalloproteinases May Influence Apoptosis W.C. Powell, L.M. Matrisian.
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Programmed cell death

Journal of Ultrastructure Research, 1974
R A, Lockshin, J, Beaulaton
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