Results 141 to 150 of about 1,462 (187)
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Veterinary Clinics of North America - Small Animal Practice, 2003
GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
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GI protein loss can result from a heterogeneous group of diseases, including lymphangiectasia, IBD, neoplasia, ulceration, intussusception, and histoplasmosis. PLE should be suspected in any hypoalbuminemic patient with no evidence of exudative protein loss, proteinuria, or HI.
Polly B, Peterson, Michael D, Willard
exaly +3 more sources
Protein-Losing Enteropathies in Dogs
Veterinary Clinics of North America - Small Animal Practice, 2011Protein-losing enteropathy is common in dogs but rare in cats. In the vast majority of cases, it is associated with inflammatory bowel disease, intestinal lymphoma, or intestinal lymphangiectasia. The diagnosis is based on identification of hypoalbuminemia and ruling out urinary loss and liver failure.
Olivier Dossin, Rachel Lavoué
exaly +3 more sources
2010
Protein-losing enteropathies are a syndrome caused by mucosal disruption or increased permeability or obstruction of lymphatic drainage. Documentation of excessive intestinal protein loss is the hallmark of the diagnosis. Treatment depends on the primary disease process and is usually a combination of dietary, medical, and/or surgical therapy.
Jonathan Goldstein, Richard Wright
exaly +2 more sources
Protein-losing enteropathies are a syndrome caused by mucosal disruption or increased permeability or obstruction of lymphatic drainage. Documentation of excessive intestinal protein loss is the hallmark of the diagnosis. Treatment depends on the primary disease process and is usually a combination of dietary, medical, and/or surgical therapy.
Jonathan Goldstein, Richard Wright
exaly +2 more sources
PROTEIN-LOSING ENTEROPATHIES IN MALIGNANCY
Annals of the New York Academy of Sciences, 1974T A, Waldmann, S, Broder, W, Strober
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Pediatric Clinics of North America, 1962
SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
+5 more sources
SummaryA man, aged 54 years, presented with œdema, ascites and hypoproteinæmia of 20 years' duration. Results of investigation of cardiac, renal and hepatic function were normal. There was no history suggesting a gastro‐intestinal disorder and investigation of bowel function was largely unproductive.
E. A. ESPINER, D. W. BEAVEN
+5 more sources
Current Opinion in Gastroenterology, 2020
Purpose of review The present review offers its readers a practical overview of protein-losing enteropathy, particularly with regard to diagnostic and therapeutic approaches. The aim is to support clinicians in their daily practice with a practical tool to deal with protein-losing enteropathy ...
Elli L., Topa M., Rimondi A.
openaire +2 more sources
Purpose of review The present review offers its readers a practical overview of protein-losing enteropathy, particularly with regard to diagnostic and therapeutic approaches. The aim is to support clinicians in their daily practice with a practical tool to deal with protein-losing enteropathy ...
Elli L., Topa M., Rimondi A.
openaire +2 more sources
Protein‐losing enteropathy in the Lundehund
Journal of Small Animal Practice, 1977ABSTRACTProtein‐losing enteropathy in the Lundehund, a possible hereditary disease, characterized clinically by intermittent diarrhoea, vomiting, wasting, ascites and oedema, is described. Serum examinations revealed low values of protein and calcium.Post‐mortem changes consisted of thickened duodenal and jejunal mucosa with blunt, short, anastomosing ...
K, Flesjå, T, Yri
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Giardiasis with Protein-Losing Enteropathy
Journal of Pediatric Gastroenterology and Nutrition, 1985Generalized edema and ascites were the main presenting features of giardiasis in a 3-year-old boy. Hypoalbuminemia, jejunal villous atrophy, Giardia lamblia in the duodenal aspirate, and abnormal gastrointestinal protein loss were present before therapy with metronidazole.
D L, Sutton, K R, Kamath
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Cardiomyopathy with Protein-Losing Enteropathy
Chest, 1973A patient with cardiomyopathy, severe hypoproteinemia and lymphocytopenia is described. Increased catabolic rate of plasma albumin and excessive intestinal protein loss were documented. In spite of the combined lymphocytopenia and hypoimmunoglobulinemia, the patient was not predisposed to infections.
I, Oliver +4 more
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Protein‐Losing Enteropathy in a Dog
Journal of Small Animal Practice, 1969Abstract— —A protein‐losing enteropathy in a dog is described. There was a history of chronic gastrointestinal upset and examination revealed hypoproteinaemic oedema and ascites. Antemortem laboratory findings indicated a protein‐losing enteropathy and these, together with the results of clinical and necropsy examination, are described.
B R, Farrow, R, Penny
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