Results 251 to 260 of about 6,504,878 (383)
Some Aspects of Family in Mahuva: A Sociological Study of Jointness in a Small Town. By L. P. Desai. Department of Sociology Publication No. 4, The M.S. University of Baroda. (New York: Asia Publishing House, 1964.) 239 pp. $7.75. [PDF]
Aileen D. Ross
openalex +1 more source
Imperial strategy of cancer cells through mitochondrial transfer
Cangkrama et al. demonstrated that cancer cells donate their mitochondria to fibroblasts through mitochondrial transfer, reprogramming them into ‘MitoCAF’. Likewise, our group has identified mitochondrial transfer from cancer cells to tumor infiltrating lymphocytes, resulting in mitochondrial ‘hijack’ and impaired antitumor immunity.
Takamasa Ishino, Yosuke Togashi
wiley +1 more source
Catch up with authors across the history of Biology Open. [PDF]
Adhikari S.
europepmc +1 more source
CDK11 inhibition stabilises the tumour suppressor p53 and triggers the production of an alternative p21WAF1 splice variant p21L, through the inactivation of the spliceosomal protein SF3B1. Unlike the canonical p21WAF1 protein, p21L is localised in the cytoplasm and has reduced cell cycle‐blocking activity.
Radovan Krejcir +12 more
wiley +1 more source
Overcoming barriers in getting research published: a commentary. [PDF]
Ravindran V.
europepmc +1 more source
I. N. Sneddon, Mixed Boundary Value Problems in Potential Theory (North-Holland Publishing Company—Amsterdam, 1966), viii+282 pp., 80s. [PDF]
J. C. Cooke
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A mouse model for vascular normalization and a human breast cancer cohort were studied to understand the relationship between vascular leakage and tumor immune suppression. For this, endothelial and immune cell RNAseq, staining for vascular function, and immune cell profiling were employed.
Liqun He +8 more
wiley +1 more source
Research That Is Not Published Is Not Worth Doing. [PDF]
Bipeta R.
europepmc +1 more source
Intein‐based modular chimeric antigen receptor platform for specific CD19/CD20 co‐targeting
CARtein is a modular CAR platform that uses split inteins to splice antigen‐recognition modules onto a universal signaling backbone, enabling precise, scarless assembly without re‐engineering signaling domains. Deployed here against CD19 and CD20 in B‐cell malignancies, the design supports flexible multi‐antigen targeting to boost T‐cell activation and
Pablo Gonzalez‐Garcia +9 more
wiley +1 more source

