Results 61 to 70 of about 158,397 (231)

Abstracts of the 3rd Rabbit Congress of the Americas [PDF]

open access: yes, 2007
not available.World Rabbit Science (2007). Abstracts of the 3rd Rabbit Congress of the Americas. World Rabbit Science. 15(1).
World Rabbit Science   +1 more
core   +1 more source

Epigenetic silencing of the liver‐specific lncRNA LUNAR promotes liver cancer progression via NOTCH activation

open access: yesMolecular Oncology, EarlyView.
LUNAR is a liver‐specific long noncoding RNA (lncRNA) that is highly expressed in normal liver but becomes epigenetically silenced in hepatocellular carcinoma through promoter hypermethylation. Loss of LUNAR is associated with NOTCH activation, epithelial–mesenchymal transition, and metastasis, whereas restoring LUNAR restrains metastatic progression ...
Se Ha Jang   +9 more
wiley   +1 more source

Abstracts of the communications presented during the 13th Hungarian Conference on Rabbit Production, Kaposvár (Hungary). May 23, 2001

open access: yes, 2002
World Rabbit Science (2002). Abstracts of the communications presented during the 13th Hungarian Conference on Rabbit Production, Kaposvár (Hungary). May 23, 2001. World Rabbit Science. 10(1).
World Rabbit Science
core   +1 more source

Abstracts of 2nd International Meeting on Rabbit Biotechnology [PDF]

open access: yes, 2008
World Rabbit Science (2008). Abstracts of 2nd International Meeting on Rabbit Biotechnology. World Rabbit Science. 16(1).
World Rabbit Science   +1 more
core   +1 more source

CEACAM1 participation in breast cancer progression

open access: yesMolecular Oncology, EarlyView.
In invasive breast cancer (BC), CEACAM1 shifts from an apical to a uniform membranous/cytoplasmic pattern, or is lost, as tumors dedifferentiate, inversely tracking the Ki‐67 proliferative index. In MCF‐7 cells, only CEACAM1‐4L suppresses proliferation, repressing cell cycle and growth factor genes.
Mykola Lyndin   +3 more
wiley   +1 more source

Epizootic rabbit enteropathy. Study of early phenomena with fresh inoculum and attempt at inactivation [PDF]

open access: yes, 2005
[EN] Using 180 35-day-old SPF rabbits, this study used the effectiveness of bacitracin as a tool for acquiring more information on the various phases of ERE, in particular during the hours inoculation.
Coudert, P., Licois, D.
core   +1 more source

Pharmacological chromatin remodeling enhances response to estrogen therapy in ER+ breast cancer

open access: yesMolecular Oncology, EarlyView.
Estrogen therapy elicits clinical benefit in ~ 30% of patients with endocrine‐resistant estrogen receptor (ER)‐positive breast cancer. Based on findings that ER transcriptional activation underlies response to estrogen therapy, we tested the effects of epigenetic dysregulation via pharmacological inhibition of histone deacetylases (HDACi).
Anneka L. Johnson Thomas   +16 more
wiley   +1 more source

Abstracts of the communications presented during the 9th French Rabbit Days.Paris, November 28-29th, 2001: NUTRITION

open access: yes, 2001
World Rabbit Science (2001). Abstracts of the communications presented during the 9th French Rabbit Days.Paris, November 28-29th, 2001: NUTRITION. World Rabbit Science. 9.
World Rabbit Science, NUTRITION,
core   +1 more source

Intrapatient tumour heterogeneity and clonal evolution in an autopsy study of metastatic salivary gland cancer

open access: yesMolecular Oncology, EarlyView.
Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche   +10 more
wiley   +1 more source

Somatostatin receptor 4 (SSTR4) is a tumor suppressor in cutaneous and head & neck squamous cell carcinomas

open access: yesMolecular Oncology, EarlyView.
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi   +6 more
wiley   +1 more source

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