Results 221 to 230 of about 224,508 (277)
Sensitivity and Hamming Graphs
ABSTRACT For any m ≥ 3 we show that the Hamming graph H ( n , m ) admits an imbalanced partition into m sets, each inducing a subgraph of low maximum degree. This improves previous results by Tandya and by Potechin and Tsang, and disproves the Strong m‐ary Sensitivity Conjecture of Asensio, García‐Marco, and Knauer.
Sara Asensio +3 more
wiley +1 more source
Correction: Investigating the role of EGFR signalling in muscle dystrophies: implications for Duchenne muscular dystrophy. [PDF]
Fernández-Simón E +9 more
europepmc +1 more source
Abstract Dystrophinopathies are caused by pathogenic variants in the DMD gene, resulting in partial (Becker) or complete loss (Duchenne) of dystrophin. Becker (BMD) and Duchenne muscular dystrophy (DMD) are characterized by progressive muscle wasting, fatty replacement, fibrosis, and loss of function.
Laura GM Heezen +14 more
wiley +1 more source
ABSTRACT Background As overall survival improves in metastatic hormone‐sensitive prostate cancer (mHSPC), non‐cancer causes of death are increasingly relevant. Methods We conducted a real‐world multicenter study of patients diagnosed with mHSPC treated with ADT alone or in combination. Causes of death before progression to castration‐resistant prostate
Marta Garcia de Herreros +15 more
wiley +1 more source
Structural Mechanism of an Efficacy Photoswitch Targeting the β<sub>2</sub>-adrenergic Receptor. [PDF]
Stipp R +29 more
europepmc +1 more source
ABSTRACT Persistent biodiversity data shortfalls undermine our capacity to detect species, map their distributions and characterize their spatial genetic structure, limiting robust biogeographic analyses and the development of effective conservation strategies.
I. Santos‐Perdomo +12 more
wiley +1 more source
Rapid generation of prion disease models using AAV‐delivered PrP variants in knockout mice
We developed a rapid AAV‐based system to generate prion disease models in weeks rather than months. Following systemic AAV9P31 delivery of modified PrP to knockout mice, we achieved brain‐wide expression and successful propagation of both classical (RML) and atypical (GSS‐A117V) prion strains.
Maitena San‐Juan‐Ansoleaga +11 more
wiley +1 more source

