Resistant hypertension and renal denervation: Who's kidding whom? [PDF]
Sever, P
core +1 more source
Consensus statement on renal denervation by the Joint Committee of Japanese Society of Hypertension (JSH), Japanese Association of Cardiovascular Intervention and Therapeutics (CVIT), and the Japanese Circulation Society (JCS) [PDF]
Kazuomi Kario +10 more
openalex +1 more source
A post‐stroke perivascular niche of microglia characterized by low expression of M2 markers and elevated glycolysis, oxidative phosphorylation (OXPHOS), and phagocytic activity is identified, which is termed stroke‐activated vascular‐associated microglia (stroke‐VAM).
Yanan Li +8 more
wiley +1 more source
Acupuncture for diabetic nephropathy: mechanisms, clinical evidence, and future perspectives. [PDF]
Yue J +7 more
europepmc +1 more source
Experimental Studies on the Nervous Control of the Renal Circulation
Jugoro Takeuchi +5 more
openalex +2 more sources
FPD‐GELNs co‐deliver Su and bioactive components. This active‐passive dual‐targeting strategy suppresses the progression of RCC through the following mechanisms: 1) inhibition of the PI3K‐Akt signaling pathway; 2) downregulation of ABCB1/P‐gp to enhance the chemosensitivity of RCC to Su; and 3) reprogramming of macrophages toward M1 polarization and ...
Haoyu Xu +8 more
wiley +1 more source
Venous congestion from a vascular waterfall perspective: reframing congestion as a dynamic Starling resistor phenomenon. [PDF]
Castro R +4 more
europepmc +1 more source
DNA Origami and Its Applications in Synthetic Biology
This review provides a comprehensive summary of recent advancements in DNA origami for synthetic biology, emphasizing two key areas: the precise construction and dynamic regulation of extracellular‐to‐intracellular, and the synergistic integration with cell‐free systems.
Yaning Fang +4 more
wiley +1 more source
Circulating Biomarkers in Failing Fontan Circulation: Current Evidence and Future Directions. [PDF]
Vecoli C, Ait-Alì L, Storti S, Foffa I.
europepmc +1 more source
NUSAP1 Recruits DAXX to Suppress HIF‐Driven Triple‐Negative Breast Cancer Progression
A double‐negative feedback loop is identified in TNBC where NUSAP1 bridges HIF and DAXX via its microtubule‐associated domain (MAD) to recruit SETDB1, repressing HIF transcriptional activity by depositing the H3K9me3 repressive mark on HIF target genes, while HIF itself suppresses NUSAP1.
Yating Du +14 more
wiley +1 more source

