Results 121 to 130 of about 6,400 (173)
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Renal Osteodystrophy

Endocrinology and Metabolism Clinics of North America, 1990
Renal osteodystrophy is a complex disorder which can be divided into five distinct bone histologic subtypes: mild bone disease, hyperparathyroid bone disease, mixed bone disease, osteomalacia, and low-turnover bone disease. Hyperparathyroidism develops in renal failure due to two principal abnormalities: 1,25(OH)2D3 deficiency and hyperphosphatemia ...
J T, McCarthy, R, Kumar
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Renal osteodystrophy

Current Opinion in Pediatrics, 1998
Secondary hyperparathyroidism develops in most patients with chronic renal failure, and is associated with the histologic finding of osteitis fibrosa cystica. The disease is characterized by growth failure and severe bone deformities in children, especially the very young.
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Diagnosis of renal osteodystrophy

European Journal of Clinical Investigation, 2006
AbstractThe idiom renal osteodystrophy (ROD) represents a heterogeneous pattern of bone disturbances caused by chronic renal insufficiency and concomitant diseases. For the clinical decision of therapy it is most important to differentiate between high and low or adynamic turnover ROD because the therapeutically consequences of these two ends of the ...
C, Schwarz, I, Sulzbacher, R, Oberbauer
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Renal osteodystrophy and hypercalcemia

Current Opinion in Rheumatology, 1993
The occurrence of aluminum-related bone disease should be completely prevented in uremic patients by restricting the use of aluminum-phosphate binders, which can be safely replaced by oral calcium carbonate. Factors other than aluminum may lead to adynamic bone disease in uremic patients.
M, Cohen-Solal, J L, Sebert
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Renale Osteodystrophie

Wiener Medizinische Wochenschrift, 2013
The incidence of renal osteodystrophy (ROD) increases with deteriorating kidney function, affecting virtually every patient on chronic dialysis treatment. ROD can persist after kidney transplantation and may be aggravated by immunosuppressants, mainly glucocorticoids.
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Aluminum and Renal Osteodystrophy

Annual Review of Medicine, 1986
Evidence has emerged over the last several years indicating that aluminum accumulation in patients with chronic renal failure can cause certain forms of renal osteodystrophy, in particular osteomalacia and an aplastic lesion. The lines of evidence include epidemiological associations, chemical measurement and histological staining of bone aluminum ...
H G, Nebeker, J W, Coburn
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Myelofibrosis and renal osteodystrophy

The American Journal of Medicine, 1977
Abstract A young patient with chronic renal failure, pancytopenia and hepatosplenomegaly with hypersplenism had evidence of severe renal osteodystrophy on x-ray films and a bone marrow biopsy specimen resembling myelofibrosis. Hematologic improvement followed splenectomy and was sustained following renal transplantation.
S G, Weinberg   +5 more
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Renal Osteodystrophy in Adults

Archives of Internal Medicine, 1956
Renal osteodystrophy may be defined as bone disease caused by chronic renal insufficiency. The skeletal changes may include osteomalacia, osteoporosis, and osteosclerosis or a combination of these. The changes may also be identical with those of osteitis fibrosa generalisata due to hyperparathyroidism. In this short paper we are not concerned with the
I M, BAIRD, F, LEES
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Bisphosphonates in renal osteodystrophy

Current Opinion in Nephrology and Hypertension, 2001
The present review considers the role that bisphosphonates might have in patients with renal failure. Although bisphosphonates are widely used to reduce fracture risk in patients with osteoporosis, few studies have documented their effect in patients with renal osteodystrophy.
S L, Fan, J, Cunningham
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Azotaemic renal osteodystrophy

Clinics in Endocrinology and Metabolism, 1957
Reviews of the natural history and pathogenesis of the osteodystrophy that develops spontaneously in the course of chronic renal failure have appeared relatively recently, both in Britain and in the U.S.A. (Kleeman et aI, 1967; Stanbury, 1967, 1968a, 1971a; Stanbury, Lumb and Mawer, 1969), and it would be unprofitable to recapitulate the details to be ...
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