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Pediatric Nephrology, 2017 Background: Hyperammonemia usually results from an inborn error of metabolism or from an advanced liver disease. Individual case reports suggest that both distal renal tubular acidosis and urinary tract infection may also result in hyperammonemia ...
Gregorio P Milani, Sebastiano A G Lava
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Dimensions of Critical Care Nursing, 2010
Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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Pregnancy and Renal Tubular Acidosis
American Journal of Perinatology, 1999Renal tubular acidosis (RTA) is uncommonly encountered in pregnancy. The risk for these women to develop pregnancy-induced hypertension has not been previously described. The renal defect noted in these women, aggravated by the normal hypervolemia of pregnancy, may predispose to hypertension.
T F, Rowe, K, Magee, F G, Cunningham
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Current Opinion in Pediatrics, 2004
Research in the past several years has led to the understanding of numerous genetic mutations that lead to inheritable forms of distal renal tubular acidosis (dRTA). Most of these mutations affect the physiology of the A-intercalated cells of the renal cortical collecting duct.
Julie A, Nicoletta, George J, Schwartz
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Research in the past several years has led to the understanding of numerous genetic mutations that lead to inheritable forms of distal renal tubular acidosis (dRTA). Most of these mutations affect the physiology of the A-intercalated cells of the renal cortical collecting duct.
Julie A, Nicoletta, George J, Schwartz
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FAMILIAL RENAL TUBULAR ACIDOSIS
Annals of Internal Medicine, 1961Excerpt Acidosis of renal origin is an increased hydrogen ion concentration due to an impairment in the excretion of titratable acid and/or ammonium ion, to an excessive excretion of bicarbonate, o...
R E, RANDALL, W H, TARGGART
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Inherited renal tubular acidosis
Current Opinion in Nephrology and Hypertension, 2000The past few years have witnessed great progress in elucidating the molecular basis of inherited renal tubular acidosis. Consistent with the physiologically defined importance of multiple gene products in urinary acidification, heritable renal tubular acidosis is genetically heterogeneous.
C, Shayakul, S L, Alper
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FAMILIAL RENAL TUBULAR ACIDOSIS
Annals of Internal Medicine, 1968Excerpt To the Editor:I was interested to read the editorial note on familial renal tubular acidosis (RTA) by Dr. Vardaman M. Buckalew (Ann. Intern. Med. 68: 1367, 1968).
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Renal Tubular Acidosis in Leprosy
Annals of Internal Medicine, 1971Excerpt To the editor: Dr. Talal's comments regarding the relationship of renal tubular acidosis to peritubular infiltration of lymphocytes (1) are of particular interest to us because of studies w...
D J, Drutz, R A, Gutman
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Evaluation of renal tubular acidosis
The Indian Journal of Pediatrics, 2007Renal tubular acidoses (RTA) comprises of a group of disorders characterized by a low capacity for net acid excretion and persistent hyperchloremic, metabolic acidosis. The RTAs are classified into chiefly three types (types 1,2 and 4) based on clinical and laboratory characteristics.
Arvind, Bagga, Aditi, Sinha
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Understanding renal tubular acidosis
British Journal of Hospital MedicineRenal tubular acidosis is a group of disorders characterised by metabolic acidosis, hyperchloraemia, normal anion gap, and potassium imbalance. Genetic mutations, drugs or acquired disorders disrupt the function of various transport proteins and enzymes in the renal tubules, causing diminished bicarbonate reabsorption or inability to excrete hydrogen ...
Samuel Sherng Young, Wang +6 more
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