Results 181 to 190 of about 20,672 (234)
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Pediatrics In Review, 2017
1. Jonathan Pelletier, MD* 2. Rasheed Gbadegesin, MD, MBBS† 3. Betty Staples, MD* 1. *Department of Pediatrics and 2. †Department of Pediatric Nephrology, Duke Children's Hospital and Health Center, Durham, NC 1. 1. Reddy P Clinical Approach to Renal Tubular Acidosis in Adult Patients. Reddy P Int J Clin Pract. 2011;65(3):350–360
Jonathan, Pelletier +2 more
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1. Jonathan Pelletier, MD* 2. Rasheed Gbadegesin, MD, MBBS† 3. Betty Staples, MD* 1. *Department of Pediatrics and 2. †Department of Pediatric Nephrology, Duke Children's Hospital and Health Center, Durham, NC 1. 1. Reddy P Clinical Approach to Renal Tubular Acidosis in Adult Patients. Reddy P Int J Clin Pract. 2011;65(3):350–360
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The Indian Journal of Pediatrics, 2015
Renal tubular acidosis (RTA) comprises a group of disorders characterized by low capacity for net acid excretion and persistent hyperchloremic metabolic acidosis, despite preserved glomerular filtration rate. RTA are classified into chiefly three types (1, 2 and 4) based on pathophysiology and clinical and laboratory characteristics. Most patients have
Arvind Bagga, Aditi Sinha
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Renal tubular acidosis (RTA) comprises a group of disorders characterized by low capacity for net acid excretion and persistent hyperchloremic metabolic acidosis, despite preserved glomerular filtration rate. RTA are classified into chiefly three types (1, 2 and 4) based on pathophysiology and clinical and laboratory characteristics. Most patients have
Arvind Bagga, Aditi Sinha
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Pediatric Nephrology, 1990
The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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Journal of Nephrology, 2006
Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Journal of Urology, 1984
Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Medical Clinics of North America, 1983
The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1999
Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Annual Review of Medicine, 1969
Renal tubular acidosis (RTA) represents a clinical syndrome characterized by a state of renal tubular insufficiency with regard to the reabsorption of bicarbonate, the excretion of net hydrogen ion, or both, and includes a large number of etiologies. In this condition, in contradistinction to the so-called glomerular acidosis, glomerular function is ...
J, Rodriguez-Soriano, C M, Edelmann
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Renal tubular acidosis (RTA) represents a clinical syndrome characterized by a state of renal tubular insufficiency with regard to the reabsorption of bicarbonate, the excretion of net hydrogen ion, or both, and includes a large number of etiologies. In this condition, in contradistinction to the so-called glomerular acidosis, glomerular function is ...
J, Rodriguez-Soriano, C M, Edelmann
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Pediatric Clinics of North America, 2019
Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Endocrinology and Metabolism Clinics of North America, 1990
Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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