Results 91 to 100 of about 185,231 (352)
Reperfusion Adjunctive Therapy: Heparin
Journal of Interventional Cardiology, 1990 Heparin therapy for patients who have undergone thrombolytic therapy of acute myocardial infarction is logical, and several trials suggest a benefit. However, in the light of the clear adjuvant benefit of aspirin, and the failure of large clinical trials to demonstrate clinically important benefits of heparin, it is not possible to make sound ...
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Advanced Science, EarlyView.This study investigates how ischemia‐free liver transplantation (IFLT) shapes both local and systemic immune responses compared with conventional liver transplantation (CLT). Our findings demonstrate that IFLT confers significant immunological protection by attenuating the STAT3‐HIF‐1α axis and enhancing the Heme‐HMOX1 pathway, two regulatory networks ...
Tao Luo +40 more
wiley +1 more source
Characteristic Signs on T2*-Based Imaging and Their Relationship with Results of Reperfusion Therapy for Acute Ischemic Stroke: A Systematic Review and Evidence to Date [PDF]
Neurointervention, 2018 Purpose Characteristic signs – the susceptibility vessel sign (SVS) and the prominent hypointense vessel sign (PHVS) – on T2*-based magnetic resonance imaging (T2*MRI) can be seen for acute ischemic stroke with large artery occlusion.
Hak Cheol Ko +5 more
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Reperfusion Therapy Starts in the Ambulance [PDF]
Circulation, 2006 Reperfusion therapy for ST-elevation acute coronary syndromes aims at early and complete recanalization of the infarct-related artery in order to salvage myocardium and improve both early and late clinical outcomes. The benefit rises exponentially the earlier therapy is initiated. The highest number of lives saved is within the first hour after symptom
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Annexin A13 Protects Against Acute Kidney Injury by Inactivating TGF‐β/Smad3 Signaling
Advanced Science, EarlyView.ANXA13 is negatively regulated by Smad3 and exerts its protective role in AKI by inactivating TGF‐β/Smad3 signaling and Smad3‐p21 cell cycle arrest pathway through binding to TβRI, inhibiting the interaction between TβRI and TβRII, thereby suppressing TβRI phosporylation.
Jiaxiao Li +12 more
wiley +1 more source
Advanced Science, EarlyView.This study identifies Rubicon as a key platelet protein that bidirectionally regulates GPVI and integrin αIIbβ3 signaling. Platelet Rubicon protects against cerebral ischemia‐reperfusion injury by limiting infarction without increasing hemorrhage.
Xiaoyan Chen +11 more
wiley +1 more source
A nationwide analysis of reperfusion therapies for pulmonary embolism in older patients with frailty [PDF]
, 2023 Ioannis T. Farmakis +9 more
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Advanced Science, EarlyView.TREM2 recognizes NETs‐derived MPO to promote DC maturation and antigen presentation, thereby exacerbating the autoimmune response in SLE. Mechanistically, TREM2 activation triggers the DAP12/SYK/ERK cascade and enhances NETs internalization by DCs, which in turn activates the cGAS/STING signaling pathway.
Jingxian Shu +7 more
wiley +1 more source
DEFECTS OF PROVIDING MEDICAL CARE TO PATIENTS WITH ST SEG-MENT ELEVATION ACUTE CORONARY SYNDROME WHO DID NOT RE-CEIVE REPERFUSION THERAPY [PDF]
, 2020 V.V. Kolosova +5 more
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Evolving Therapies for Myocardial Ischemia/Reperfusion Injury
Journal of the American College of Cardiology, 2015 The damage inflicted on the myocardium during acute myocardial infarction is the result of 2 processes: ischemia and subsequent reperfusion (ischemia/reperfusion injury). During the last 3 decades, therapies to reduce ischemic injury (mainly reperfusion strategies) have been widely incorporated into clinical practice.
Ibanez, B. +3 more
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