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Soluble IL-2 receptor and memory Treg profiles differentiate early rheumatoid arthritis from undifferentiated arthritis at initial presentation. [PDF]
Zi X, Shi Y, Zhao Y, Gao C, Wang C.
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Causal association between rheumatoid arthritis and periodontitis: A Mendelian randomization study. [PDF]
Li X, Ouyang Y, Liang L, Li Y.
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Long-term complete radiological response to immunotherapy in a patient with metastatic NSCLC and active rheumatoid arthritis. [PDF]
Roy E +3 more
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T Cell Clones in Rheumatoid Arthritis (RA)
Scandinavian Journal of Rheumatology, 1988The development of T cell clones has greatly enhanced our knowledge of pathogenetic mechanisms in autoimmune diseases. Thus, it has been possible to induce experimental arthritis in susceptible animals by solely injecting arthritogenic T cell lines or clones.
N, Hain, G R, Burmester, J R, Kalden
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RETRACTED: Rheumatoid arthritis (RA) and cardiovascular disease
Autoimmunity Reviews, 2019Abstract Patients with rheumatoid arthritis (RA) suffer cardiovascular events 1.5-2 fold than the general population, and cardiovascular (CV) events are leading cause of death in patients with RA. It is known that patients with RA have endothelial dysfunction, related with impaired function of endothelial progenitor cells (EPCs).
Arnon, Blum, Mohammad, Adawi
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[Genetics in rheumatoid arthritis (RA)].
Polski merkuriusz lekarski : organ Polskiego Towarzystwa Lekarskiego, 2007Rheumatoid arthritis (RA) is a chronic autoimmune disease. Genetic and environmental factors are implicated in the pathogenesis of RA. In this study we describe numerous genetic phenomena that may be implicated in the etiopathogenesis of RA i.e. antigens of major histocompatibility complex, genetic linkage analysis results of whole genome scan and ...
Jadwiga, Skretkowicz +1 more
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The role of the endothelium in rheumatoid arthritis (RA)
Pathology, 1992RA is a systemic disease characterised by inflammation and destruction of synovial joints. The synovial microvasculature undergoes two distinct morphological changes - namely the formation of high endothelial venules (HEVs) and the development of new blood vessels (angiogenesis or neovascularisation) which contribute to joint inflammation and ...
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