Results 311 to 320 of about 917,291 (368)
Some of the next articles are maybe not open access.

Treatment of complex I deficiency with riboflavin

Journal of the Neurological Sciences, 1993
We have evaluated the effects of treatment with riboflavin in five patients with a mitochondrial myopathy, associated with a complex I (NADH dehydrogenase) deficiency. Two patients suffered from a clinically pure myopathy and the other patients presented with encephalomyopathic features.
Wim Ruitenbeek   +3 more
openaire   +3 more sources

Dietary riboflavin deficiency induces ariboflavinosis and esophageal epithelial atrophy in association with modification of gut microbiota in rats

European Journal of Nutrition, 2020
Feng Pan   +8 more
semanticscholar   +1 more source

Riboflavin Deficiency in Man

Nutrition Reviews, 2009
W. H. Sebrell, R. E. Butler
openaire   +4 more sources

INTERSTITIAL KERATITIS AND RIBOFLAVIN DEFICIENCY

Archives of Ophthalmology, 1940
To the Editor: —A letter by Dr. L. V. Johnson and Mr. R. E. Eckardt was published in the March number of theArchivesunder the heading "Is the Onset of Interstitial Keratitis Related to Riboflavin Deficiency?" (page 631). This letter criticizes us for the wording of a news release published in the Cleveland Press of Feb.
V. P. Sydenstricker   +3 more
openaire   +2 more sources

Clinical Significance of Riboflavin Deficiency

1980
The clinical consequences of riboflavin deficiency in humans were first described by Sebrell and Butler (1938). On a diet which furnished only 0.5 mg of riboflavin daily, 10 of 18 adult women developed oral lesions that disappeared following riboflavin supplementation.
Surat Komindr, George Nichoalds
openaire   +2 more sources

Riboflavin Deficiency in the Lamb

Journal of Animal Science, 1950
R. Culik   +4 more
openaire   +3 more sources

Riboflavin transporter deficiency

Neuromuscular Disorders, 2017
Haliloglu, G.   +4 more
openaire   +3 more sources

Metabolism of Pyridoxine in Riboflavin Deficiency [PDF]

open access: possible, 1980
Epithelial lesions of the mouth have been reported to respond to treatment with either riboflavin or pyridoxine. Earlier we had hypothesized that these lesions may be due to cellular deficiency of pyridoxal phosphate (PLP), since the enzyme pyridoxaminephosphate oxidase (PPO) is a flavo protein.
openaire   +1 more source

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