Results 311 to 320 of about 310,561 (353)
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Right Ventricular Failure and Pulmonary Hypertension

Emergency Medicine Clinics of North America, 2022
Right ventricular dysfunction is an important component of the pathophysiology of several disorders commonly encountered in the emergency department (ED). Interventions often performed routinely early in the ED course such as fluid administration and endotracheal intubation have the potential to cause precipitous clinical deterioration in patients with
Sara E, Crager, Caroline, Humphreys
openaire   +2 more sources

Right Ventricular Failure After Left Ventricular Assist Device

Cardiology Clinics, 2020
Right ventricular failure after left ventricular assist device (LVAD) implantation remains common in the contemporary, continuous-flow era. Clinically meaningful, reproducible, and consensus definitions of both early and late right ventricular failure after LVAD are needed for progress in advanced heart failure.
Rebecca, Cogswell   +2 more
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Postoperative Right Ventricular Failure

2020
This chapter examines postoperative right ventricular (RV) failure. RV failure is not uncommon following cardiac surgery and often portends a worse clinical outcome. After establishing the diagnosis of RV failure, the central goal of treatment is to increase RV function in order to achieve adequate left ventricular preload and cardiac output.
Marguerite Hoyler, Natalia S. Ivascu
openaire   +1 more source

Right ventricular hemangioma causing right ventricular inflow obstruction and right heart failure

Journal of the American Society of Echocardiography, 2004
We report a patient who presented with symptoms of right heart failure. Transesophageal echocardiography revealed a right ventricular mass, causing right ventricular inflow obstruction. Coronary angiography revealed a characteristic tumor blush. After successful surgical resection, histologic findings were consistent with a right ventricular hemangioma.
Heidar, Arjomand   +5 more
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Physiopathology of Right Ventricular Failure

Seminars in Thoracic and Cardiovascular Surgery: Pediatric Cardiac Surgery Annual, 2006
The physiopathology of right ventricular failure after repair of tetralogy of Fallot is complex. While primarily reflecting chronic volume overload resulting from pulmonary regurgitation, its determinants reflect a varied contribution of different boundary conditions.
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Pathophysiology of right ventricular failure

Critical Care Medicine, 2008
Right ventricular failure may be defined as the inability of the right ventricle of the heart to provide adequate blood flow through the pulmonary circulation at a normal central venous pressure. Critical care specialists encounter right ventricular failure routinely in their practice, but until recently right ventricular failure as a primary clinical ...
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Right Ventricular Failure

New England Journal of Medicine, 2023
Brian A. Houston   +2 more
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Acute Right Ventricular Failure

2014
Acute right ventricular (RV) failure is a devastating syndrome caused by a variety of common diseases and conditions. Acute RV failure is caused by acute alterations in preload, afterload, and/or contractility. Ventricular interdependence and decreases in perfusion pressure make the RV particularly prone to acute failure.
Anthony R. Cucci   +2 more
openaire   +1 more source

Right Ventricular Failure: Pathophysiology and Treatment

Surgical Clinics of North America, 1985
The cardiac surgeon is faced with RV failure in two main situations: in isolation or in patients with left-sided cardiac assist. Adequate volume loading, correction of acidosis and oxygenation, cardiac pacing, pharmacologic agents, and systemic intra-aortic balloon pumping allow stabilization in most of these patients.
P A, Spence, R D, Weisel, T A, Salerno
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Right ventricular failure associated with left ventricular failure.

Cardiovascular clinics, 1987
RV and LV failure frequently coexist. Experimental evidence suggests that RV failure results from a primary insult to the right ventricle and an increase in pulmonary vascular resistance. LV failure results in an elevation of the left atrial pressure and thereby a reduction in the transpulmonary hydrostatic gradient.
P A, Spence   +3 more
openaire   +1 more source

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