Results 71 to 80 of about 88,528 (305)

Spamming in scholarly publishing: A case study [PDF]

open access: yesJournal of the Association for Information Science and Technology, 2015
Spam has become an issue of concern in almost all areas where the Internet is involved, and many people today have become victims of spam from publishers and individual journals. We studied this phenomenon in the field of scholarly publishing from the perspective of a single author.
Marcin Kozak   +2 more
openaire   +1 more source

Toolkits for Equity : transforming scholarly publishing communities : guidelines on inclusive language and images in scholarly communication [PDF]

open access: yes, 2022
Comprend des références bibliographiques et webographiquesToolkits for Equity Project"The Guidelines on Inclusive Language and Images in Scholarly Communication are a global tool, educational resource, and living archive to help all authors, editors, and

core  

AscF in the mycobacterial CIII–CIV supercomplex lacks metal and nucleotide binding but links malate oxidation to respiration

open access: yesFEBS Letters, EarlyView.
The mycobacterial CIII‐CIV respiratory supercomplex is an obligate assembly, encompassing several subunits of unknown functions. We have characterized the intracellular subunit AscF, and show that it is unlikely to be a sensor for metals or nucleotides, but is required for growth on nonfermentable energy sources, and likely works as an adapter for ...
Eni Rile   +8 more
wiley   +1 more source

The Future of Pediatric Hematology‐Oncology Fellowship Training: Reflections on the American Board of Pediatrics Training Model

open access: yes
Pediatric Blood &Cancer, EarlyView.
Scott C. Borinstein   +11 more
wiley   +1 more source

CCDC80 suppresses high‐grade serous ovarian cancer migration via negative regulation of B7‐H3

open access: yesMolecular Oncology, EarlyView.
PAX8 is a lineage‐specific master regulator of transcription in high‐grade serous ovarian cancer (HGSC) progression. We show for the first time that PAX8 facilitates proliferation and metastasis by repressing the cell autonomous tumor suppressor CCDC80 and inducing B7‐H3 expression.
Aya Saleh   +12 more
wiley   +1 more source

New Gateways to Scholarly Communication through Open Access [PDF]

open access: yes, 2010
The draumatic changes in 21st century has been occurred in the world of publication of scolarly communication.One of the phenomenais of Open Access Publishing Model.The open access movement is increasingly guiding the publishing practices of scholarly ...
Pradip, Joshi, Nikose, S.M.
core  

Latest trends in innovative global scholarly journal publication and distribution platforms [PDF]

open access: yesScience Editing, 2018
This review article presents the latest trends in innovative global scholarly journal publication and distribution platforms, with implications for local journals.
Soon Kim, Eunkyung Chung, Jae Yun Lee
doaj   +1 more source

Oncogenic DMTF1β promotes cancer cell motility by regulating autophagy through ULK1 stabilization

open access: yesMolecular Oncology, EarlyView.
In the current study, we demonstrate that the oncogene DMTF1β regulates ULK1 stability by reducing its proteasomal degradation in cancer cells. This stabilization enables ULK1 to induce autophagy, which in turn facilitates cancer cell migration. Consequently, reduced DMTF1β levels lead to decreased autophagy and impaired cancer cell migration.
Jun Xu   +13 more
wiley   +1 more source

Scholarly communication in networked environment: problems and prospects [PDF]

open access: yes, 2007
This article gives an overview of how popularity of World Wide Web and open access concepts challenge the existence of traditional scholarly communication.
Vimal Kumar, V.
core  

Systemic dysregulation of apolipoproteins in amyotrophic lateral sclerosis serum

open access: yesFEBS Open Bio, EarlyView.
Amyotrophic lateral sclerosis (ALS) is a fatal disease that damages motor neurons. This study found that people with ALS show significant changes in blood fats and the proteins that carry them. Several apolipoproteins were higher, lipid balances were altered, and normal protein–lipid relationships were disrupted.
Finula I. Isik   +6 more
wiley   +1 more source

Home - About - Disclaimer - Privacy