Results 241 to 250 of about 11,085 (277)

Causes of DNA mismatch repair deficiency in sebaceous skin lesions demonstrating loss of MLH1 protein expression: constitutional over somatic MLH1 promoter methylation. [PDF]

open access: yesFam Cancer
Joo JE   +9 more
europepmc   +1 more source

Topics in histopathology of sweat gland and sebaceous neoplasms

The Journal of Dermatology, 2017
AbstractThis article reviews several topics regarding sweat gland and sebaceous neoplasms. First, the clinicopathological characteristics of poroid neoplasms are summarized. It was recently reported that one‐fourth of poroid neoplasms are composite tumors and one‐fourth are apocrine type lesions.
openaire   +2 more sources

[Torre-Muir syndrome. Sebaceous gland neoplasms, keratoacanthomas, multiple internal cancers and heredity].

Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete, 1982
The Torre-Muir syndrome includes multiple sebaceous neoplasms and keratoacanthomas, as well as multiple internal malignancies. We describe three cases of this unusual association which has not previously been reported in the German-speaking literature. The Torre-Muir syndrome appears to have a close connection to the cancer family syndrome [17, 34].
W I, Worret   +3 more
openaire   +1 more source

Diagnostic Accuracy of Immunohistochemistry Testing on Sebaceous Gland Neoplasms for Muir‐Torre Syndrome: A Meta‐Analysis

Journal of Cutaneous Pathology
ABSTRACT Background Muir‐Torre syndrome (MTS) is a hereditary tumor predisposition syndrome associated with sebaceous neoplasms. Immunohistochemistry (IHC) for loss of mismatch repair (MMR) proteins in these tumors is used as a screening test, but its diagnostic accuracy has not been rigorously
Jack Hulse   +3 more
openaire   +1 more source

Preferential expression of OVOL1 in inner root sheath of hair, sebaceous gland, eccrine duct and their neoplasms in human skin.

Fukuoka igaku zasshi = Hukuoka acta medica, 2015
OVOL1 is an important transcription factor for epidermal keratinization, which suppresses proliferation and switches on the differentiation of keratinocytes. A recent genome-wide association study has revealed that OVOL1 is one of the genes associated with susceptibility to atopic dermatitis. Although it is known to be expressed in murine skin and hair
Chikage, Mitoma   +18 more
openaire   +1 more source

132: Sebaceous Gland Carcinoma: A Capricious Neoplasm

Otolaryngology–Head and Neck Surgery, 1996
Seth P. Linker, K. Thomas Robbins
openaire   +1 more source

Cytologic Features of Sebaceous Neoplasms of the Parotid Gland: Sebaceous Lymphadenoma and Sebaceous Carcinoma

Journal of the American Society of Cytopathology, 2019
Maryna Vazmitsel   +2 more
openaire   +1 more source

The contemporary management of cancers of the sinonasal tract in adults

Ca-A Cancer Journal for Clinicians, 2023
Rajat Thawani
exaly  

Abundant expression of neuronatin in normal eccrine, apocrine and sebaceous glands and their neoplasms

The Journal of Dermatology, 2010
Long, Dugu   +5 more
openaire   +2 more sources

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