Results 181 to 190 of about 2,720,543 (229)
Some of the next articles are maybe not open access.
Secondary and Tertiary Hyperparathyroidism
Journal of Clinical Densitometry, 2013We reviewed the etiology and management of secondary and tertiary hyperparathyroidism. Secondary hyperparathyroidism is characterized by an increase in parathyroid hormone (PTH) that is appropriate and in response to a stimulus, most commonly low serum calcium. In secondary hyperparathyroidism, the serum calcium is normal and the PTH level is elevated.
Sophie A Jamal
exaly +3 more sources
Secondary and Tertiary Hyperparathyroidism
, 2018 Secondary hyperparathyroidism (SHP) is a frequent complication of kidney diseases. At variance with all the other forms of SHP, which are compensatory conditions, renal SHP has many pathogenetic peculiarities, which have been only in part defined.
Piergiorgio, Messa, Carlo Maria, Alfieri
openaire +3 more sources
Otolaryngologic Clinics of North America
Secondary hyperparathyroidism (SHPT) does not initiate as a primary dysfunction of parathyroid glands resulting from an intrinsic defect or disease but is the physiologic response of parathyroids to metabolic changes elsewhere in the body occurring over time.
Palak Choksi, Krupa Doshi
openaire +3 more sources
Secondary hyperparathyroidism (SHPT) does not initiate as a primary dysfunction of parathyroid glands resulting from an intrinsic defect or disease but is the physiologic response of parathyroids to metabolic changes elsewhere in the body occurring over time.
Palak Choksi, Krupa Doshi
openaire +3 more sources
Mechanisms of secondary hyperparathyroidism
American Journal of Physiology-Renal Physiology, 2002Small decreases in serum Ca2+and more prolonged increases in serum phosphate (Pi) stimulate the parathyroid (PT) to secrete parathyroid hormone (PTH), and 1,25(OH)2D3decreases PTH synthesis and secretion. A prolonged decrease in serum Ca2+and 1,25(OH)2D3, or increase in serum Pi, such as in patients with chronic renal failure, leads to the appropriate ...
Justin, Silver +2 more
openaire +2 more sources
Cimetidine in Secondary Hyperparathyroidism
Annals of Internal Medicine, 1982Excerpt To the editor: After anecdotal reports on a decrease of immunoreactive paraphyroid hormone (PTH) levels due to the administration of cimetidine in primary hyperparathyroidism (1,2), therape...
J, Kovarik +3 more
openaire +2 more sources
Myelofibrosis Secondary to Hyperparathyroidism
Experimental and Clinical Endocrinology & Diabetes, 2004We report on a young female who had presented with fatigue, bilateral knee pain and gait disturbance. Primary hyperparathyroidism was diagnosed together with splenomegaly and anemia. Bone marrow biopsy revealed myelofibrosis. A parathyroid adenoma was excised during surgical intervention.
B, Kumbasar +5 more
openaire +2 more sources
Secondary hypercalcemic hyperparathyroidism
Virchows Archiv Abteilung A Pathologische Anatomie, 1973A search was made for patients with associated secondary hyperparathyroidism and hypercalcemia: 22 such cases were found in the literature and 22 were recorded among 92 patients operated upon because of parathyroid disease. In the remaining 70 patients the effect of the operation on the serum calcium level was investigated: persisting hypercalcemia ...
L, Bergdahl, L, Boquist
openaire +2 more sources
[Secondary hyperparathyroidism].
Chirurgia italiana, 2001We reviewed the results of twenty-four years' experience of parathyroidectomy for secondary hyperparathyroidism. Over the period from 1976 to 2000, we performed 171 parathyroidectomies for secondary hyperparathyroidism (91 males, 80 females; median age: 57.4). Autotransplantation was performed in 45. Of the 171 patients undergoing parathyroidectomy, 14
SIANESI M. +3 more
openaire +4 more sources
Management of Secondary Hyperparathyroidism
Therapeutic Apheresis and Dialysis, 2005Abstract: Secondary hyperparathyroidism (SHPT) remains an inevitable consequence of untreated chronic uremia. It is the result of a combination of phosphate (P) retention, failure of calcitriol synthesis, and hypocalcemia. Therapies used to correct these abnormalities, namely active vitamin D replacement, calcium (Ca) supplementation, and phosphate (P)
openaire +3 more sources
The prevention of secondary hyperparathyroidism
Current Opinion in Nephrology and Hypertension, 1993It has become increasingly evident that clear-cut disturbances of bone and mineral metabolism develop early in renal failure. Among these disturbances, hyperparathyroidism is well documented and is usually asymptomatic at that early stage. It is now accepted that early therapy using phosphate restriction, through diet and calcium-containing phosphate ...
openaire +2 more sources