Results 241 to 250 of about 47,738 (289)
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Otolaryngologic Clinics of North America
Secondary hyperparathyroidism (SHPT) does not initiate as a primary dysfunction of parathyroid glands resulting from an intrinsic defect or disease but is the physiologic response of parathyroids to metabolic changes elsewhere in the body occurring over time.
Palak Choksi, Krupa Doshi
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Secondary hyperparathyroidism (SHPT) does not initiate as a primary dysfunction of parathyroid glands resulting from an intrinsic defect or disease but is the physiologic response of parathyroids to metabolic changes elsewhere in the body occurring over time.
Palak Choksi, Krupa Doshi
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Management of Secondary Hyperparathyroidism
Therapeutic Apheresis and Dialysis, 2005Abstract: Secondary hyperparathyroidism (SHPT) remains an inevitable consequence of untreated chronic uremia. It is the result of a combination of phosphate (P) retention, failure of calcitriol synthesis, and hypocalcemia. Therapies used to correct these abnormalities, namely active vitamin D replacement, calcium (Ca) supplementation, and phosphate (P)
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[Secondary hyperparathyroidism].
Deutsche medizinische Wochenschrift (1946), 2001We reviewed the results of twenty-four years' experience of parathyroidectomy for secondary hyperparathyroidism. Over the period from 1976 to 2000, we performed 171 parathyroidectomies for secondary hyperparathyroidism (91 males, 80 females; median age: 57.4). Autotransplantation was performed in 45. Of the 171 patients undergoing parathyroidectomy, 14
SIANESI M. +3 more
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DeckerMed Complex General Surgical Oncology, 2019
Secondary hyperparathyroidism is defined and its pathophysiology, delineated. Key components of the diagnostic work-up, medical management, and indications for surgery are described. The operative approach and controversy on extent of parathyroidectomy are discussed. This review contains 3 figures, 1 tables, and 24 references.
Sonia L Sugg, Anna C Beck
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Secondary hyperparathyroidism is defined and its pathophysiology, delineated. Key components of the diagnostic work-up, medical management, and indications for surgery are described. The operative approach and controversy on extent of parathyroidectomy are discussed. This review contains 3 figures, 1 tables, and 24 references.
Sonia L Sugg, Anna C Beck
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Mechanisms of secondary hyperparathyroidism
American Journal of Physiology-Renal Physiology, 2002Small decreases in serum Ca2+and more prolonged increases in serum phosphate (Pi) stimulate the parathyroid (PT) to secrete parathyroid hormone (PTH), and 1,25(OH)2D3decreases PTH synthesis and secretion. A prolonged decrease in serum Ca2+and 1,25(OH)2D3, or increase in serum Pi, such as in patients with chronic renal failure, leads to the appropriate ...
Justin, Silver +2 more
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Hyperparathyroidism Secondary to Hyperaldosteronism
High Blood Pressure & Cardiovascular Prevention, 2010Introduction: Hyperparathyroidism has been associated with essential hypertension. In rats, volume expansion, the deoxycorticosterone (DOC)-salt hypertension model and primary aldosteronism cause a reduction in plasma calcium levels and secondary hyperparathyroidism. In these rats, urinary calcium decreases with thiazides and/or aldosterone antagonists,
Fortina F., Bellosta S.
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Myelofibrosis Secondary to Hyperparathyroidism
Experimental and Clinical Endocrinology & Diabetes, 2004We report on a young female who had presented with fatigue, bilateral knee pain and gait disturbance. Primary hyperparathyroidism was diagnosed together with splenomegaly and anemia. Bone marrow biopsy revealed myelofibrosis. A parathyroid adenoma was excised during surgical intervention.
B, Kumbasar +5 more
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Secondary and Tertiary Hyperparathyroidism
Journal of Clinical Densitometry, 2013We reviewed the etiology and management of secondary and tertiary hyperparathyroidism. Secondary hyperparathyroidism is characterized by an increase in parathyroid hormone (PTH) that is appropriate and in response to a stimulus, most commonly low serum calcium. In secondary hyperparathyroidism, the serum calcium is normal and the PTH level is elevated.
Sophie A, Jamal, Paul D, Miller
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Cimetidine in Secondary Hyperparathyroidism
Annals of Internal Medicine, 1982Excerpt To the editor: After anecdotal reports on a decrease of immunoreactive paraphyroid hormone (PTH) levels due to the administration of cimetidine in primary hyperparathyroidism (1,2), therape...
J, Kovarik +3 more
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Secondary and Tertiary Hyperparathyroidism
2018Secondary hyperparathyroidism (SHP) is a frequent complication of kidney diseases. At variance with all the other forms of SHP, which are compensatory conditions, renal SHP has many pathogenetic peculiarities, which have been only in part defined.
Messa, Piergiorgio, Alfieri, Carlo Maria
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