Results 281 to 290 of about 2,760,769 (327)
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The prevention of secondary hyperparathyroidism
Current Opinion in Nephrology and Hypertension, 1993It has become increasingly evident that clear-cut disturbances of bone and mineral metabolism develop early in renal failure. Among these disturbances, hyperparathyroidism is well documented and is usually asymptomatic at that early stage. It is now accepted that early therapy using phosphate restriction, through diet and calcium-containing phosphate ...
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Anemia and Secondary Hyperparathyroidism
Archives of Internal Medicine, 1980To the Editor. —The article entitled "Anemia and Secondary Hyperparathyroidism" in the NovemberArchives(138:1650-1652, 1978) reports that subtotal parathyroidectomy resulted in a substantial increase of mean hematocrit value and hemoglobin level, but the precise mechanism was unknown. The reason the mechanism was unknown is because the authors did not
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Renal transplantation and secondary hyperparathyroidism
Seminars in Surgical Oncology, 1997Secondary hyperparathyroidism (2'HPT) improves after renal transplantation (RTx) along with recovered function of the renal allograft. However, normal renal function does not last long due to rejection, drug-induced nephrotoxic nepthropathy, or recurrence of post-transplant glomerulonephritis.
Hiroshi Takagi +3 more
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Secondary Hyperparathyroidism: A Case Report
Journal of Endodontics, 2003A thorough diagnostic examination is essential before providing endodontic treatment. The sequence of diagnostic procedures must begin with a well-organized review of the medical history. In the early screening process, a health history that reveals a systemic disorder must be investigated further because it may have a significant impact on the dental ...
Robert J. Loushine +3 more
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Secondary hyperparathyroidism complicated by parathyromatosis
American Journal of Kidney Diseases, 1996Secondary hyperparathyroidism is a common complication of chronic renal disease. Clinical signs and symptoms tend to be severe and often are not controlled with medical measures. When medical therapy fails, parathyroidectomy becomes necessary. Recurrent hyperparathyroidism is not uncommon following surgery.
Donald J. Sherrard +7 more
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[Secondary hyperparathyroidism].
Chirurgia italiana, 2001We reviewed the results of twenty-four years' experience of parathyroidectomy for secondary hyperparathyroidism. Over the period from 1976 to 2000, we performed 171 parathyroidectomies for secondary hyperparathyroidism (91 males, 80 females; median age: 57.4). Autotransplantation was performed in 45. Of the 171 patients undergoing parathyroidectomy, 14
SIANESI M. +3 more
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Renal transplant and secondary hyperparathyroidism
Scandinavian Journal of Urology and Nephrology, 2007To investigate the impact of mineral metabolism, renal function, months on dialysis and months since transplant for predicting intact parathyroid hormone (iPTH) levels in a cohort of patients who had undergone their first renal graft with estimated glomerular filtration rates (eGFRs) of 30-60 and >60 ml/min/1.73 m2.One hundred and twenty-eight patients
Lasse G. Gøransson +2 more
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Surgical management of secondary hyperparathyroidism
Advances in Renal Replacement Therapy, 2002Most patients with renal failure maintained on chronic dialysis have elevated parathyroid hormone (PTH) levels and PTH-mediated bone disease (secondary hyperparathyroidism [sHPT]). Elevated PTH production in this setting represents a progressive, exaggerated physiologic response to hypocalcemia by the parathyroid glands, and generalized growth of the ...
George S. Leight, John A. Olson
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Brown Tumor and Secondary Hyperparathyroidism
Archives of Ophthalmology, 1991Brown tumor is a focal, bony lesion of hyperparathyroidism that results from parathyroid hormone on bone increasing osteoclastic activity with bone resorption and trabecular fibrosis. This leads to microfractures and hemorrhage and the appearance of brown tumors, which are seen most commonly in primary hyperparathyroidism and less frequently in ...
Alfredo E. Chu +2 more
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Neuromuscular Disease in Secondary Hyperparathyroidism
Annals of Internal Medicine, 1975Neuromuscular function was evaluated in six patients with osteomalacia or secondary hyperparathyroidism, or both, as demonstrated by bone biopsy showing osteomalacia or increased immunoreactive parathyroid hormone, or both. Each patient had weakness, atrophy, and fatigability of proximal muscles, especially of the lower extremities.
L. E. Mallette +2 more
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