Soluble suppression of tumorigenicity 2 is a potential predictor of post-liver transplant renal outcomes [PDF]
Acute kidney injury is considered an independent prognostic factor for mortality in patients with liver cirrhosis. Non-treated acute kidney injury can progress to hepatorenal syndrome with a poor prognosis. As suppression of tumorigenicity 2 (ST2) is a member of the interleukin-1 receptor family that aggravates inflammation and fibrotic changes in ...
Jong Joo Moon +12 more
doaj +5 more sources
Soluble suppression of tumorigenicity 2 associated with atrial fibrillation detected after stroke: A retrospective study [PDF]
Background: The soluble suppression of tumorigenicity 2 (sST2) is closely associated with stroke and atrial fibrillation (AF). However, no studies on sST2 and AF detected after stroke (AFDAS) have been reported.
Wenquan Hou +6 more
doaj +6 more sources
Evaluation of soluble suppression of tumorigenicity 2 (sST2) as serum marker for liver fibrosis [PDF]
Background & aims With the increase in patients at risk of advanced liver disease due to the obesity epidemic, there will be a need for simple screening tools for advanced liver fibrosis.
Florian F. Hildenbrand +8 more
doaj +7 more sources
The soluble suppression of tumorigenicity 2 as a biomarker of early cardiac remodeling in bradycardia patients receiving permanent pacemaker therapy [PDF]
Aim: This study aims to evaluate: the difference of soluble suppression of tumorigenicity 2 (sST2) level, a biomarker for cardiac remodeling and echocardiography parameters value prior to and 1 month after implantation; and the association between ...
Mohammad Iqbal +6 more
doaj +5 more sources
Predictive value of soluble suppression of tumorigenicity 2 in atrial fibrillation: a systematic review and meta-analysis [PDF]
PurposeAtrial fibrosis is the main pathological basis for the pathogenesis and progression of atrial fibrillation (AF). Soluble suppression of tumorigenicity 2 (sST2) is involved in fibrosis.
Pengfei Chen +8 more
doaj +4 more sources
Soluble Suppression of Tumorigenicity-2 Associates With Ventilator Dependence in Coronavirus Disease 2019 Respiratory Failure [PDF]
OBJECTIVES:. We hypothesize that elevated soluble suppression of tumorigenicity-2 concentrations, a marker of pulmonary epithelial injury, reflect ongoing lung injury in acute hypoxemic respiratory failure due to coronavirus disease 2019 and associate ...
Jehan W. Alladina, MD +10 more
doaj +4 more sources
Soluble suppression of tumorigenicity 2 associated with major adverse cardiac events in children with myocarditis [PDF]
ObjectiveSoluble suppression of tumorigenicity 2 (sST2) is associated with the prognosis of some cardiac diseases, but studies on sST2 and the prognosis of patients with myocarditis are rare. This study investigated the relationship between major adverse
Tongtong Shi +3 more
doaj +4 more sources
Serum soluble suppression of tumorigenicity-2 level associates with severity of pulmonary hypertension associated with uncorrected atrial septal defect [PDF]
Uncorrected atrial septal defect undergoes right ventricle chronic volume overload which may lead to pulmonary hypertension and Eisenmenger Syndrome. The soluble suppression of tumorigenicity-2 is a left ventricle strain biomarker; however, its role in ...
Reza S. Pratama +4 more
doaj +3 more sources
Soluble suppression of tumorigenicity 2 as outcome predictor after cardiopulmonary resuscitation: an observational prospective study [PDF]
Prognostication after cardiopulmonary resuscitation (CPR) is complex. Novel biomarkers like soluble suppression of tumorigenicity 2 (sST2) may provide an objective approach.
Richard Rezar +13 more
doaj +5 more sources
Soluble suppression of tumorigenicity 2 associated with microvascular obstruction in patients with ST-segment elevation myocardial infarction [PDF]
Background Microvascular obstruction (MVO) develops in approximately 50% of patients with ST-segment elevation myocardial infarction (STEMI) after undergoing percutaneous coronary intervention (PCI).
Xinjia Du +6 more
doaj +4 more sources

