Results 121 to 130 of about 1,398 (171)
Effect of growth hormone on colonic anastomosis after intraperitoneal administration of 5-fluorouracil, bleomycin and cisplatin: An experimental study. [PDF]
World J Gastrointest SurgLambrou I +15 more
europepmc +1 more source
Impact of Gestational Diabetes Mellitus on Fetal Growth and Nutritional Status in Newborns. [PDF]
NutrientsKarcz K, Królak-Olejnik B.
europepmc +1 more source
Some of the next articles are maybe not open access.
Nutrition and somatomedin XVI: Somatomedins and somatomedin inhibitors in fasted and refed rats
Metabolism: Clinical and Experimental, 1988Nutritional deprivation is associated with poor growth and decreased levels of net circulating somatomedin activity, as measured by bioassay. Since somatomedin activity reflects the contributions both of somatomedins (which stimulate cartilage) and of somatomedin inhibitors (which antagonize the ability of the somatomedins to stimulate cartilage), we ...
L S Phillips +2 more
exaly +3 more sources
Clinical Orthopaedics and Related Research, 1975
Somatomedin is a peptide component of serum which has been postulated to mediate the action of growth hormone on skeletal tissue. Direct effects on cartilage include stimulation of the synthesis of mucopolysacharide, protein, and nucleic acids. Insulin-like effects on non-skeletal tissues and cells have been described, and a relationship to NSILA-S and
J D, Bomboy, W D, Salmon
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Somatomedin is a peptide component of serum which has been postulated to mediate the action of growth hormone on skeletal tissue. Direct effects on cartilage include stimulation of the synthesis of mucopolysacharide, protein, and nucleic acids. Insulin-like effects on non-skeletal tissues and cells have been described, and a relationship to NSILA-S and
J D, Bomboy, W D, Salmon
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International Journal of Biochemistry & Cell Biology, 1985
Bioassayable somatomedins and somatomedin inhibitors were examined after chromatographic separation, using serum from normal rats (enriched in somatomedins) and diabetic rats (enriched in somatomedin inhibitors). At neutral pH, gel filtration on Sephacryl S-300 revealed somatomedins at mol.
Lawrence S Phillips, S Goldstein
exaly +3 more sources
Bioassayable somatomedins and somatomedin inhibitors were examined after chromatographic separation, using serum from normal rats (enriched in somatomedins) and diabetic rats (enriched in somatomedin inhibitors). At neutral pH, gel filtration on Sephacryl S-300 revealed somatomedins at mol.
Lawrence S Phillips, S Goldstein
exaly +3 more sources
Hormone Research, 2008
Somatomedin levels measured by radioreceptor assay, competitive protein-binding assay or radioimmunoassay are normal in hypercortisolism; the decrease of somatomedin activity consistently found in this condition is due to an increase in circulating somatomedin inhibitors resulting in an inhibition of somatomedin action. Progestagens could possibly have
Caufriez, Anne, Copinschi, Georges
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Somatomedin levels measured by radioreceptor assay, competitive protein-binding assay or radioimmunoassay are normal in hypercortisolism; the decrease of somatomedin activity consistently found in this condition is due to an increase in circulating somatomedin inhibitors resulting in an inhibition of somatomedin action. Progestagens could possibly have
Caufriez, Anne, Copinschi, Georges
openaire +3 more sources
Glucocorticoid Effects on Somatomedins and Somatomedin Inhibitors*
The Journal of Clinical Endocrinology & Metabolism, 1985Glucocorticoid excess may be associated with poor growth despite normal levels of GH and adequate nutrition. Steroid-induced growth failure could be mediated by defective generation and/or action of somatomedins. To probe potential mechanisms, we examined the effect of corticosteroid administration on net somatomedin activity, immunoreactive ...
T G, Unterman, L S, Phillips
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Normal Somatomedin and Somatomedin Receptors in Achondroplastic Dwarfism
Hormone and Metabolic Research, 1980It has been proposed that the basic abnormality in achondroplasia may be a quantitative defect in endochondral new bone formation secondary to decreased synthesis of somatomedin (SM) or abnormal binding of SM to specific receptors. To test this hypothesis, we have measured plasma SM levels and SM receptors on circulating mononuclear cells obtained from
R G, Rosenfeld, R L, Hintz
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