Results 311 to 320 of about 121,621 (353)
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Neurology, 1995
There you go again: Th steadfast fad of fixing spasticity. To the Editor: Presentation of the problem. The Role of tizanidine in the treatment of spasticity, the recent 80-page supplement to Neurology [1] subsidized by the manufacturer of tizanidine, sets me up front [2] as the stalking horse entree of the symposium. Of course, I was flattered to gain
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There you go again: Th steadfast fad of fixing spasticity. To the Editor: Presentation of the problem. The Role of tizanidine in the treatment of spasticity, the recent 80-page supplement to Neurology [1] subsidized by the manufacturer of tizanidine, sets me up front [2] as the stalking horse entree of the symposium. Of course, I was flattered to gain
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IEEE Transactions on Biomedical Engineering, 1989
A model of hemiplegic spasticity based on electromyographical and biomechanical parameters measured during passive muscle stretching is presented. Two components of spasticity can be distinguished--phasic and tonic. This classification depends on the pattern of stretch reflex activity which can be either phasic or tonic as well as on the muscle stretch/
Stefanovska, A. +4 more
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A model of hemiplegic spasticity based on electromyographical and biomechanical parameters measured during passive muscle stretching is presented. Two components of spasticity can be distinguished--phasic and tonic. This classification depends on the pattern of stretch reflex activity which can be either phasic or tonic as well as on the muscle stretch/
Stefanovska, A. +4 more
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American Journal of Physical Medicine & Rehabilitation, 1988
The functional impairment due to spasticity must be carefully assessed before any treatment is considered. Therapeutic intervention is best individualized to a particular patient. Basic principles of treatment to ameliorate spastic hypertonia are: 1) avoid noxious stimuli and 2) provide frequent range of motion.
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The functional impairment due to spasticity must be carefully assessed before any treatment is considered. Therapeutic intervention is best individualized to a particular patient. Basic principles of treatment to ameliorate spastic hypertonia are: 1) avoid noxious stimuli and 2) provide frequent range of motion.
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Current Opinion in Neurology, 1997
Recent open studies and two placebo-controlled studies confirm the potential role of Botulinum toxin A in the treatment of focal spasticity in adults and children. The effect of the toxin might not only be mediated by the paresis of extrafusal, but also intrafusal muscle fibres, thereby altering the afferent discharge.
Karl-Heinz Mauritz, Stefan Hesse
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Recent open studies and two placebo-controlled studies confirm the potential role of Botulinum toxin A in the treatment of focal spasticity in adults and children. The effect of the toxin might not only be mediated by the paresis of extrafusal, but also intrafusal muscle fibres, thereby altering the afferent discharge.
Karl-Heinz Mauritz, Stefan Hesse
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2023
Spasticity is characterized by an enhanced size and reduced threshold for activation of stretch reflexes and is associated with "positive signs" such as clonus and spasms, as well as "negative features" such as paresis and a loss of automatic postural responses.
Jonathan, Marsden +2 more
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Spasticity is characterized by an enhanced size and reduced threshold for activation of stretch reflexes and is associated with "positive signs" such as clonus and spasms, as well as "negative features" such as paresis and a loss of automatic postural responses.
Jonathan, Marsden +2 more
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Electroencephalography and Clinical Neurophysiology, 1983
Many different methods have been used to assess spasticity. They are all based on measurements of the responses to muscle stretch. The methods differ in how muscle stretch is obtained and how the responses are measured.
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Many different methods have been used to assess spasticity. They are all based on measurements of the responses to muscle stretch. The methods differ in how muscle stretch is obtained and how the responses are measured.
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The pathophysiology of spasticity
European Journal of Neurology, 2002Spasticity is only one of several components of the upper motor neurone (UMN) syndrome, known collectively as the `positive' phenomena, that are characterized by muscle overactivity. Other components include tendon hyper‐reflexia, clonus, the clasp‐knife phenomenon, flexor and extensor spasms, a Babinski sign, and spastic dystonia. Spasticity is a form
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Developmental Medicine & Child Neurology, 1966
SUMMARYIn placing a cerebral palsied person in water, the supine position is generally preferable to the prone, although supination may stimulate extensor spasm. Holding the breath is preferable to blowing out as it requires less concentration and can be maintained for a longer time.
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SUMMARYIn placing a cerebral palsied person in water, the supine position is generally preferable to the prone, although supination may stimulate extensor spasm. Holding the breath is preferable to blowing out as it requires less concentration and can be maintained for a longer time.
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Spasticity and `Spastic' Gait in Children with Cerebral palsy
Neuroscience & Biobehavioral Reviews, 1998The current notion of spasticity as a velocity-dependent increase of muscle response to imposed stretch was mainly derived from studies performed under stationary experimental conditions. To address the issue of a spastic muscle behaviour under dynamic conditions, we conceived a novel approach, aimed at quantitatively assessing motor output over the ...
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