Results 341 to 350 of about 164,048 (382)
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Spasticity management in the child with spastic quadriplegia
European Journal of Neurology, 2001In children with spastic quadriplegia, also described as ‘whole body involvement’, spasticity can interfere with motor function, contributes to the development of deformities and adversely impacts on care, positioning, and comfort. In this population, spasticity interventions address goals such as improving comfort, reducing pain, easing the burden of ...
M E, Gormley, L E, Krach, L, Piccini
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International Journal of Neuroscience, 2018
Purpose/aim: To evaluate the efficacy of tetrahydrocannabinol (THC):cannabidiol (CBD) oromucosal spray (Sativex®) as add-on therapy to optimised standard antispasticity treatment in patients with moderate to severe multiple sclerosis (MS) spasticity ...
J. Marková +6 more
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Purpose/aim: To evaluate the efficacy of tetrahydrocannabinol (THC):cannabidiol (CBD) oromucosal spray (Sativex®) as add-on therapy to optimised standard antispasticity treatment in patients with moderate to severe multiple sclerosis (MS) spasticity ...
J. Marková +6 more
semanticscholar +1 more source
Stereotactic and Functional Neurosurgery, 2000
Spasticity is usually a useful substitute for deficiency of motor strength. However not infrequently, it may become harmful leading to an aggravation of motor disability. When excessive spasticity is not sufficiently controlled by physical therapy and pharmacological treatment, patients can have recourse to neurosurgery: neurostimulation, intrathecal ...
M P, Sindou, P, Mertens
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Spasticity is usually a useful substitute for deficiency of motor strength. However not infrequently, it may become harmful leading to an aggravation of motor disability. When excessive spasticity is not sufficiently controlled by physical therapy and pharmacological treatment, patients can have recourse to neurosurgery: neurostimulation, intrathecal ...
M P, Sindou, P, Mertens
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Neurology, 1995
There you go again: Th steadfast fad of fixing spasticity. To the Editor: Presentation of the problem. The Role of tizanidine in the treatment of spasticity, the recent 80-page supplement to Neurology [1] subsidized by the manufacturer of tizanidine, sets me up front [2] as the stalking horse entree of the symposium. Of course, I was flattered to gain
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There you go again: Th steadfast fad of fixing spasticity. To the Editor: Presentation of the problem. The Role of tizanidine in the treatment of spasticity, the recent 80-page supplement to Neurology [1] subsidized by the manufacturer of tizanidine, sets me up front [2] as the stalking horse entree of the symposium. Of course, I was flattered to gain
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American Journal of Physical Medicine & Rehabilitation, 1988
The functional impairment due to spasticity must be carefully assessed before any treatment is considered. Therapeutic intervention is best individualized to a particular patient. Basic principles of treatment to ameliorate spastic hypertonia are: 1) avoid noxious stimuli and 2) provide frequent range of motion.
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The functional impairment due to spasticity must be carefully assessed before any treatment is considered. Therapeutic intervention is best individualized to a particular patient. Basic principles of treatment to ameliorate spastic hypertonia are: 1) avoid noxious stimuli and 2) provide frequent range of motion.
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Annals of Otology, Rhinology & Laryngology, 1976
130 cases of spastic dysphonia are reviewed with special emphasis on the etiology of the disorder. An analysis of the circumstances surrounding the onset of the disease points strongly to a psychogenic origin of the disease in, at least, the vast majority of the cases.
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130 cases of spastic dysphonia are reviewed with special emphasis on the etiology of the disorder. An analysis of the circumstances surrounding the onset of the disease points strongly to a psychogenic origin of the disease in, at least, the vast majority of the cases.
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IEEE Transactions on Biomedical Engineering, 1989
A model of hemiplegic spasticity based on electromyographical and biomechanical parameters measured during passive muscle stretching is presented. Two components of spasticity can be distinguished--phasic and tonic. This classification depends on the pattern of stretch reflex activity which can be either phasic or tonic as well as on the muscle stretch/
Stefanovska, A. +4 more
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A model of hemiplegic spasticity based on electromyographical and biomechanical parameters measured during passive muscle stretching is presented. Two components of spasticity can be distinguished--phasic and tonic. This classification depends on the pattern of stretch reflex activity which can be either phasic or tonic as well as on the muscle stretch/
Stefanovska, A. +4 more
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Drug and Therapeutics Bulletin, 2000
Spasticity is a frequent feature of neurological disease in adults and children. Commonly associated symptoms include impaired movement, painful muscle spasms, stiffness and disturbed sleep. Severe or long-standing spasticity may lead to contractures and joint ankylosis, which can severely restrict the patient's care and rehabilitation. Here, we review
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Spasticity is a frequent feature of neurological disease in adults and children. Commonly associated symptoms include impaired movement, painful muscle spasms, stiffness and disturbed sleep. Severe or long-standing spasticity may lead to contractures and joint ankylosis, which can severely restrict the patient's care and rehabilitation. Here, we review
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Current Opinion in Neurology, 1997
Recent open studies and two placebo-controlled studies confirm the potential role of Botulinum toxin A in the treatment of focal spasticity in adults and children. The effect of the toxin might not only be mediated by the paresis of extrafusal, but also intrafusal muscle fibres, thereby altering the afferent discharge.
S, Hesse, K H, Mauritz
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Recent open studies and two placebo-controlled studies confirm the potential role of Botulinum toxin A in the treatment of focal spasticity in adults and children. The effect of the toxin might not only be mediated by the paresis of extrafusal, but also intrafusal muscle fibres, thereby altering the afferent discharge.
S, Hesse, K H, Mauritz
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The pathophysiology of spasticity
European Journal of Neurology, 2002Spasticity is only one of several components of the upper motor neurone (UMN) syndrome, known collectively as the `positive' phenomena, that are characterized by muscle overactivity. Other components include tendon hyper‐reflexia, clonus, the clasp‐knife phenomenon, flexor and extensor spasms, a Babinski sign, and spastic dystonia. Spasticity is a form
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