Results 121 to 130 of about 341,259 (167)

Controlled trial of thyrotropin releasing hormone tartrate in ataxia of spinocerebellar degenerations

Journal of the Neurological Sciences, 1983
Tetsuya Takayanagi, T Nakanishi, Tadao Tsubaki   +2 more
exaly   +2 more sources

Cerebral blood flow in spinocerebellar degenerations: a single photon emission tomography study in 28 patients

Journal of Neurology, 1998
Andrea Soricelli, Elena Salvatore, A Postiglione   +2 more
exaly   +2 more sources

Bedside and electro‐oculographic analysis of abnormal ocular movements in spinocerebellar degenerations

Neurology, 1988
Itsuro Sobue
exaly   +2 more sources

Simple and clear differentiation of spinocerebellar degenerations: Overview of macroscopic and low‐power view findings

Neuropathology (Kyoto. 1993), 2022
Spinocerebellar degenerations (SCDs) are a diverse group of rare and slowly progressive neurological diseases that include spinocerebellar ataxia type 1 (SCA1), SCA2, SCA3, SCA6, SCA7, dentatorubral‐pallidoluysian atrophy (DRPLA) and multiple system ...
K. Iwabuchi, S. Koyano, S. Yagishita
semanticscholar   +1 more source

Normal muscle pyruvate oxidation in spinocerebellar degenerations

Annals of Neurology, 1981
Owen B Evans
exaly   +2 more sources

Adrenoleukodystrophy Presenting as Spinocerebellar Degeneration

European Neurology, 2008
The clinical features of 3 patients from a kindred with adrenoleukodystrophy and the analysis of their plasma sphingomyelin are described. Onset of symptoms was between the ages of 33 and 54 years. Ataxic gait and spasticity were the only symptoms noted during the early stage of the disorder. Dementia and optic atrophy were present in two of the cases.
T, Nakazato, T, Sato, T, Nakamura, S, Tsuji, H, Narabayashi   +4 more
openaire   +2 more sources

Slow Saccades in Spinocerebellar Degeneration

Archives of Neurology, 1976
Two patients with spinocerebellar degeneration made abnormally slow horizontal refixations. One patient produced quick phases of nystagmus with identical maximum velocities, suggesting her refixations were abnormal saccades and not voluntary pursuit movements.
D S, Zee, L M, Optican, J D, Cook, D A, Robinson, W K, Engel   +4 more
openaire   +2 more sources

Spinocerebellar Degeneration

Journal of Computer Assisted Tomography, 1988
Magnetic resonance imaging at 1.5 T was performed in 27 patients with either the cerebellar or spinocerebellar form of spinocerebellar degeneration and in 10 control subjects. Neither T1- nor T2-weighted images (T1WIs and T2WIs) of the patients showed any abnormal intensity areas within the cerebellum or in any other structures of the brain.
H, Nabatame, H, Fukuyama, I, Akiguchi, M, Kameyama, K, Nishimura, Y, Nakano   +5 more
openaire   +2 more sources

Adrenoleukomyelo‐neuropathy presenting as spinocerebellar degeneration

Neurology, 1982
A spinocerebellar syndrome has been described rarely in adrenoleukodystrophy. We report a family containing three cases of typical adrenoleukodystrophy, and one patient with a slowly progressive spinocerebellar syndrome. A high index of suspicion and the ACTH stimulation test are required when dealing with multisystem neurologic disease of unknown ...
C D, Marsden, J A, Obeso, A E, Lang
openaire   +2 more sources

[Treatment for Spinocerebellar Degeneration].

Brain and nerve = Shinkei kenkyu no shinpo, 2023
No disease-modifying therapy has been established for spinocerebellar degeneration and multiple system atrophy, and only symptomatic therapy is currently available. Taltirelin and protirelin are drugs covered by health insurance for cerebellar ataxia symptoms, and are expected to suppress the progression of symptoms.
Masaaki, Matsushima, Ichiro, Yabe
openaire   +1 more source