Results 101 to 110 of about 185,380 (253)
2015 Annual report, Alice Springs Steiner School
Made available via the Publications (Legal Deposit) Act 2004 (NT)
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Alice Springs Steiner School newsletter
This publication contains many links to external sites. These external sites may no longer be active.Made available via the Publications (Legal Deposit) Act 2004 (NT)
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Somatic mutational landscape in von Hippel–Lindau familial hemangioblastoma
The causes of central nervous system (CNS) hemangioblastoma in Von Hippel–Lindau (vHL) disease are unclear. We used Whole Exome Sequencing (WES) on familial hemangioblastoma to investigate events that underlie tumor development. Our findings suggest that VHL loss creates a permissive environment for tumor formation, while additional alterations ...
Maja Dembic +5 more
wiley +1 more source
Alice Springs Town Council annual report 2000-2001
Made available by the Northern Territory Library via the Publications (Legal Deposit) Act 2004 (NT)
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CCDC80 suppresses high‐grade serous ovarian cancer migration via negative regulation of B7‐H3
PAX8 is a lineage‐specific master regulator of transcription in high‐grade serous ovarian cancer (HGSC) progression. We show for the first time that PAX8 facilitates proliferation and metastasis by repressing the cell autonomous tumor suppressor CCDC80 and inducing B7‐H3 expression.
Aya Saleh +12 more
wiley +1 more source
Alice Springs Town Council annual report 2010/2011
Made available by the Northern Territory Library via the Publications (Legal Deposit) Act 2004 (NT)
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KDM7A and KDM1A inhibition suppresses tumour promoting pathways in prostate cancer
Treatment resistance is a major challenge for patients with advanced prostate cancer. This study examined an alternative approach to target the major prostate cancer‐promoting pathway by targeting epigenetic factors, whose levels are higher in tumours.
Jennie N Jeyapalan +16 more
wiley +1 more source
Alice Springs Town Council 2018-2021 strategic plan
Made available by the Northern Territory Library via the Publications (Legal Deposit) Act 2004 (NT)
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We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu +10 more
wiley +1 more source
Alice Springs Town Council annual report 2012-2013
Made available by the Northern Territory Library via the Publications (Legal Deposit) Act 2004 (NT)
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