CASUAL AND BASAL BLOOD PRESSURES IV. THEIR RELATIONSHIP TO THE SUPPLEMENTAL PRESSURE WITH A NOTE ON STATISTICAL IMPLICATIONS [PDF]
, 1944 F. H. Smirk
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Molecular Oncology, EarlyView.We investigated whether hypomethylating agents (HMAs) used in myeloid malignancies induce somatic retrotransposition. Our findings indicate that HMA treatment increases L1‐encoded protein expression but does not lead to detectable de novo retrotransposition events in either patient samples or cell lines.
Šárka Pavlová +14 more
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ImMLPro platform for accessible machine learning and statistical analysis in digital agriculture and beyond. [PDF]
Sci RepJeelani MI, Mansoor S.
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P-Technique: A New Statistical Device for Analysing Functional Unities in the Intact Organism [PDF]
, 1953 Raymond B. Cattell, H. F. V. M. Williams
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A new statistical method for haplotype reconstruction from population data.
American Journal of Human Genetics, 2001 M. Stephens, N. J. Smith, P. Donnelly
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YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma
Molecular Oncology, EarlyView.The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy +9 more
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Computer vision based efficient segmentation and classification of multi brain tumor using computed tomography images. [PDF]
Sci RepAli A +4 more
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Emerging role of ARHGAP29 in melanoma cell phenotype switching
Molecular Oncology, EarlyView.This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
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Mendelian randomization analysis reveals no causal association between non-alcoholic fatty liver disease and hepatocellular carcinoma: implications for lipid metabolomics and shared pathophysiological mechanisms. [PDF]
Discov OncolWei Y, Li J.
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