Results 281 to 290 of about 79,534 (300)
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Growth of Streptococcus mutans in a chemostat
Archives of Oral Biology, 1974Abstract A strain of Streptococcus mutans isolated from a carious lesion of a patient and known to cause caries in hamsters and monkeys were grown in a complex medium in a chemostat. There were changes in the ability of the organism to stick to surfaces, glucose utilization, and acid production of the organism when grown at different dilution rates.
D. C. Ellwood+2 more
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Empyema due to Streptococcus mutans
Chest, 1977Empyema due to Streptococcus mutans occurred following dental manipulation in two patients with periodontal disease. Isolation of this dental pathogen from pleural fluid localized the site of origin of the empyemas to the oropharynx and precluded the need to search for a remote intra-abdominal source of these pleuropulmonary infections.
Joel Ruskin+2 more
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Recurrent streptococcus mutans endocarditis
The American Journal of Medicine, 1987Recurrent bacterial endocarditis is associated with several risk factors including parenteral drug abuse, past valvular heart disease, and periodontitis. Recurrence rates vary from 2 to 18 percent for a single recurrence, and 1 to 5 percent for two recurrences.
Philip W. Smith+6 more
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Lipid Composition of Streptococcus mutans
Caries Research, 1978Cellular fatty acids and extractable intact lipids of five strains of Streptococcus mutans grown on chemically defined medium were studied. The fatty acids were characterized by the predominance of the C16:0 and C18:1 components as well as traces of cyclopropane acids.
E.I. Szabo, B.H. Amdur, S.S. Socransky
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Cariogenicity of Mutants of Streptococcus mutans
Journal of Dental Research, 1980In this study, we induced a number of mutants of S. mutans differing in their abilities to synthesize glucan and produce acid. When tested for cariogenicity in gnotobiotic rats, "high pH" mutants caused less caries, but could not supplant a cariogenic parent strain.
S. Rosen, Michael W.H. Mao
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Pulp response to Streptococcus mutans
Oral Surgery, Oral Medicine, Oral Pathology, 1987The maxillary molar pulps of germ-free rats were mechanically exposed, and suspensions of a strain of freshly grown Streptococcus mutans were applied to the pulp wounds. The pulps were left open to the oral environment, and the animals were maintained in the isolator until they were killed in groups after 2, 7, and 28 days.
Robert C. Paterson, Stephen K. Pountney
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Chromosomal Deletions in Streptococcus mutans
1997The oral bacterium Streptococcus mutans possesses the ability to ferment a wide range of carbohydrates, which results in the production of acids that can cause demineralisation of tooth enamel and subsequent dental caries. However, it has been shown that approximately 11% of independent isolates of S.
Roy R. B. Russell, C. R. Lewis
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Invertase activity in Streptococcus mutans and Streptococcus sanguis
Archives of Oral Biology, 1973Abstract Toluene treatment of intact cells revealed invertase-like activity in Streptococcus mutans K1-R (cariogenic) and in Streptococcus sanguis 903–1600 (noncariogenic). The activity was identified as a β- d -fructofuranoside fructohydrolase (invertase; EC.
M.M. McCabe, R.A. Cowman, E.E. Smith
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A Levansucrase from Streptococcus mutans
Caries Research, 1970One of the predominant bacteria in early dental plaque, Streptococcus mutans , has been shown to synthesize levansucrase.
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Virulence properties of Streptococcus Mutans
Frontiers in Bioscience, 2004Streptococcus mutans is considered one of the primary causative agents of dental caries and can also be a source of infective endocarditis. The main virulence factors associated with cariogenicity include adhesion, acidogenicity, and acid tolerance. Each of these properties works coordinately to alter dental plaque ecology.
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