Results 41 to 50 of about 147,775 (297)

Stress-related cardiomyopathies [PDF]

open access: yesAnnals of Intensive Care, 2011
Stress-related cardiomyopathies can be observed in the four following situations: Takotsubo cardiomyopathy or apical ballooning syndrome; acute left ventricular dysfunction associated with subarachnoid hemorrhage; acute left ventricular dysfunction associated with pheochromocytoma and exogenous catecholamine administration; acute left ventricular ...
openaire   +2 more sources

Stress-induced cardiomyopathy associated with ipratropium bromide therapy in a patient with chronic obstructive pulmonary disease

open access: yesRevista Portuguesa de Cardiologia, 2014
Stress-induced cardiomyopathy, also known as ‘broken heart syndrome’ or Takotsubo cardiomyopathy, is characterized by transient systolic dysfunction of the apical and/or mid segments of the left ventricle, in the absence of significant coronary artery ...
Filipa Melão   +8 more
doaj   +1 more source

Triggers for atrial fibrillation. the role of anxiety [PDF]

open access: yes, 2019
Atrial fibrillation (AF) is the most widely recognized arrhythmia. Systemic arterial hypertension, diabetes, obesity, heart failure, and valvular heart diseases are major risk factors for the onset and progression of AF.
Ceccacci, Andrea   +10 more
core   +1 more source

Stress-induced cardiomyopathy

open access: yesCurrent Cardiovascular Imaging Reports, 2009
Stress-induced cardiomyopathy is a relatively uncommon syndrome with difficult definition. Cases with transient and reversible left ventricular dysfunction, precipitated by emotional stress and without coronary artery disease, are called takotsubo syndrome, left ventricular apical ballooning, and broken heart syndrome.
Ronderos, Ricardo E.   +2 more
openaire   +2 more sources

TakoTsubo Syndrome: A Well-Known Disease but Not Everything Is Clear Yet

open access: yesReviews in Cardiovascular Medicine, 2022
TakoTsubo Syndrome (TTS) is a stress-induced cardiac disease characterized by temporary and segmental left ventricle dysfunction, typically involving the apex. Post-menopause women are more frequently affected.
Cesare de Gregorio   +5 more
doaj   +1 more source

Enhanced cardiac expression of two isoforms of matrix metalloproteinase-2 in experimental diabetes mellitus. [PDF]

open access: yes, 2019
BackgroundDiabetic cardiomyopathy (DM CMP) is defined as cardiomyocyte damage and ventricular dysfunction directly associated with diabetes independent of concomitant coronary artery disease or hypertension.
Ahn, Jinhee   +15 more
core   +2 more sources

Echocardiography in the Diagnosis of Cardiomyopathies: Current Status and Future Directions

open access: yesReviews in Cardiovascular Medicine, 2022
Cardiomyopathies are a challenging pathology and echocardiography is essential for diagnosis and prognosis. The most frequent cardiomyopathies are the dilated cardiomyopathy (DCM) and the hypertrophic cardiomyopathy (HCM), followed by the less frequent ...
Livia Trasca   +3 more
doaj   +1 more source

Adenine Nucleotide Translocase 1 Expression is Coupled to the HSP27-Mediated TLR4 Signaling in Cardiomyocytes [PDF]

open access: yes, 2019
The cardiac-specific overexpression of the adenine nucleotide translocase 1 (ANT1) has cardioprotective effects in various experimental heart disease models.
Dörner, Andrea   +6 more
core   +2 more sources

Stress-Induced Cardiomyopathy: As a Diagnosis That Is Time Sensitive and Anticipative in Certain Individuals

open access: yesCase Reports in Cardiology, 2018
Stress-induced cardiomyopathy, also known as Takotsubo cardiomyopathy, presents as an acute coronary syndrome. However, the physiology and pathogenesis are different.
Vishal Dhulipala   +3 more
doaj   +1 more source

Metabolic and cardiac adaptation to chronic pharmacologic blockade of facilitative glucose transport in murine dilated cardiomyopathy and myocardial ischemia [PDF]

open access: yes, 2018
GLUT transgenic and knockout mice have provided valuable insight into the role of facilitative glucose transporters (GLUTs) in cardiovascular and metabolic disease, but compensatory physiological changes can hinder interpretation of these models.
Heitmeier, Monique R.   +6 more
core   +2 more sources

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