Results 11 to 20 of about 1,739 (160)

Rapid detection of avian leukosis virus subgroup J by cross-priming amplification [PDF]

open access: yesScientific Reports, 2021
Avian leukosis virus subgroup J (ALV-J) causes oncogenic disease in chickens in China, resulting in great harm to poultry production, and remains widespread in China.
Yong Xiang   +8 more
doaj   +4 more sources

Novel mutation of avian leukosis virus subgroup J from Tibetan chickens

open access: yesPoultry Science, 2021
Tibetan chickens are descendants of the ancestral red jungle fowl Gallus gallus. Very little is known about pathogens in Tibetan chickens living in the high-altitude environment.
Moru Xu   +8 more
doaj   +3 more sources

Diversity of Avian leukosis virus subgroup J in local chickens, Jiangxi, China [PDF]

open access: yesScientific Reports, 2021
Avian leukosis caused by avian leukosis virus (ALV) is one of the most severe diseases endangering the poultry industry. When the eradication measures performed in commercial broilers and layers have achieved excellent results, ALV in some local chickens
Haiqin Li   +10 more
doaj   +3 more sources

Avian Leukosis Virus Subgroup J in Layer Chickens, China

open access: yesEmerging Infectious Diseases, 2010
To the Editor: In recent years, cases of avian leukosis virus subgroup J (ALV-J) infection and tumors in commercial layer chickens and breeders of egg-type chickens have been emerging in the People’s Republic of China. ALV-J was first isolated from meat-type chickens with myeloid leukosis in 1988.
Yu-Long Gao   +6 more
doaj   +3 more sources

Research Progress on Pathogenesis and Prevention of Avian Leukosis Virus J Subgroup (ALV-J) [PDF]

open access: yesVeterinary Sciences
As a major retrovirus threatening global poultry farming, Avian Leukosis Virus Subgroup J (ALV-J) has expanded its host range since discovery, extending from conventional broilers to layer chickens and native breeds. Its diverse oncogenic manifestations,
Xinyu Liu, Xi Lan
doaj   +2 more sources

Enhanced inhibition of Avian leukosis virus subgroup J replication by multi-target miRNAs [PDF]

open access: yesVirology Journal, 2011
Background Avian leukosis virus (ALV) is a major infectious disease that impacts the poultry industry worldwide. Despite intensive efforts, no effective vaccine has been developed against ALV because of mutations that lead to resistant forms.
Meng Qing-Wen   +4 more
doaj   +3 more sources

Mapping the mutational landscape of an avian retrovirus envelope protein across its evolutionary trajectory. [PDF]

open access: yesPLoS Pathogens
Many RNA viruses exhibit error-prone replication. Continuous generation of erroneous copies accelerates evolution. Avian leukosis virus subgroup J (ALV-J), an avian oncogenic virus, is a classical model virus for studying retroviruses.
Moru Xu   +8 more
doaj   +2 more sources

Reticuloendotheliosis virus and avian leukosis virus subgroup J synergistically increase the accumulation of exosomal miRNAs [PDF]

open access: yesRetrovirology, 2018
Background Co-infection with avian leukosis virus subgroup J and reticuloendotheliosis virus induces synergistic pathogenic effects and increases mortality.
Defang Zhou   +9 more
doaj   +3 more sources

Role of O-linked glycosylation modification on internalization and replication of avian leukosis virus subgroup J [PDF]

open access: yesVeterinary Research
Upon infection, viruses reprogram the host metabolic system to hijack metabolic resources for proliferation. Avian leukosis virus subgroup J (ALV-J), an avian oncogenic virus, poses significant challenges to the poultry industry. ALV-J infection upgrades
Moru Xu   +7 more
doaj   +2 more sources

Semen extracellular vesicles mediate vertical transmission of subgroup J avian leukosis virus. [PDF]

open access: yesVirol Sin, 2022
Subgroup J avian leukosis virus (ALV-J) is a highly oncogenic retrovirus that has been devastating the global poultry industry since the late 1990s. The major infection model of ALV-J is vertical transmission, which is responsible for the congenital infection of progeny from generation to generation. Increasing evidence has suggested that extracellular
Liao L   +11 more
europepmc   +3 more sources

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