Results 231 to 240 of about 382,582 (334)

Clinical Presentations, Biochemical Phenotypes, and Genotype-Phenotype Correlations in Patients withSuccinate Dehydrogenase Subunit B-Associated Pheochromocytomas and Paragangliomas [PDF]

open access: bronze, 2007
Henri Timmers   +7 more
openalex   +1 more source

Succinate dehydrogenase inhibition with malonate during reperfusion reduces infarct size by preventing mitochondrial permeability transition.

open access: yesCardiovascular Research, 2016
Laura Valls-Lacalle   +7 more
semanticscholar   +1 more source

Cell Size and Succinate Dehydrogenase Activity of Different Types of Fibers in Different Regions of the Tibialis Anterior Muscle in Mice and Rats.

open access: bronze, 2000
Chiyoko Hirofuji   +7 more
openalex   +2 more sources

Mitochondrial alterations as potential early events in neuromuscular junction remodelling with muscle disuse

open access: yesExperimental Physiology, EarlyView.
Abstract Physical inactivity impairs neuromuscular health, promoting skeletal muscle atrophy, mitochondrial changes, and neuromuscular junction (NMJ) instability. However, the interplay between mitochondria and NMJ alterations in the context of muscle disuse remains underexplored.
Evgeniia Motanova   +6 more
wiley   +1 more source

Itaconate drives mtRNA-mediated type I interferon production through inhibition of succinate dehydrogenase. [PDF]

open access: yesNat Metab
O'Carroll SM   +15 more
europepmc   +1 more source

A distinctive, low‐grade oncocytic fumarate hydratase‐deficient renal cell carcinoma, morphologically reminiscent of succinate dehydrogenase‐deficient renal cell carcinoma

open access: yesHistopathology, 2017
Steven C. Smith   +16 more
semanticscholar   +1 more source

Sex‐dependent effects of a high‐fat diet‐induced obesity model on cerebrovascular health and brain metabolism

open access: yesExperimental Physiology, EarlyView.
Abstract Mid‐life obesity is a major risk factor for neurodegenerative diseases, with mitochondrial and cerebrovascular dysfunction considered key mediators. Lysine acetylation is a reversible post‐translational modification that regulates several mitochondrial metabolic and biochemical processes.
Nicole N. Eminhizer   +11 more
wiley   +1 more source

Dietary nitrate supplementation mitigates age‐related changes at the neuromuscular junction in mice

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Ageing is associated with a decline in muscle mass and strength, namely sarcopenia, significantly impacting the quality of life in older individuals. Different strategies are being explored to counteract these detrimental effects. In the present study, we investigated a non‐invasive nutritional approach: the impact of 2 months of
Maira Rossi   +9 more
wiley   +1 more source

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