Results 151 to 160 of about 4,185 (189)

Masked bilateral superior oblique palsy

Canadian Journal of Ophthalmology, 2003
A diagnosis of masked bilateral superior oblique palsy (MBSOP) is established when signs of SOP appear in the normal eye of a patient after strabismus surgery for SOP in the contralateral eye. Despite the absence of signs of bilaterality before surgery, a palsy will develop in the previously unaffected eye in 10% or more of the patients undergoing ...
Faisal, Esmail, Michael, Flanders
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Acquired Superior Oblique Palsy

Archives of Ophthalmology, 1967
In 40 cases of acquired superior oblique palsy, head trauma was the most frequent cause. The pattern of the resulting vertical, horizontal, and torsional defects is discussed. Data are presented, indicating that the habitual head tilt is a compensation to reduce the vertical deviation, and thus aid fusion.
E, Khawam, A B, Scott, A, Jampolsky
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Benign Palsy of Superior Oblique Muscle

Journal of Pediatric Ophthalmology & Strabismus, 1980
Thirteen patients with nontraumatic superior oblique muscle palsy were examined during a four to seven year follow-up period. Extensive investigations disclosed no underlying systemic or neurologic diseases. In all patients a quick improvement and recovery of the motility imbalance occurred spontaneously in about 10 weeks, suggesting a benign prognosis
P, Nemet, V, Godel, E, Baruch, M, Lazar
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Tendon Laxity in Superior Oblique Palsy

Ophthalmology, 1992
Some congenital superior oblique palsies are known to be due to anomalous or lax superior oblique tendons. This prospective study of 24 patients with a diagnosis of superior oblique palsy was performed to determine the prevalence and significance of anatomic tendon anomaly or laxity as diagnosed by traction testing of the superior oblique tendon ...
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Acquired bilateral superior oblique palsy

Documenta Ophthalmologica, 1973
Acquired bilateral superior oblique palsy following severe head injury is characterized by a disabling torsional diplopia, with a right hypertropia present in gaze down left, and a left hypertropia present in gaze down right. It is caused by a hemorrhage in the roof of the midbrain at the caudal end of the quadrigeminal plate.
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Traction Testing in Superior Oblique Palsy

Journal of Pediatric Ophthalmology & Strabismus, 1990
ABSTRACT This report describes the use of superior oblique traction testing in the evaluation of superior oblique palsy. Four consecutive children presenting with signs and symptoms of superior oblique palsy were found to have markedly abnormal tendon laxity with traction testing.
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Classification and Treatment of Superior Oblique Palsy

American Orthoptic Journal, 1974
(1974). Classification and Treatment of Superior Oblique Palsy. American Orthoptic Journal: Vol. 24, No. 1, pp. 18-22.
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Isolated Superior Oblique Tucking: An Effective Procedure for Superior Oblique Palsy with Profound Superior Oblique Underaction

Journal of American Association for Pediatric Ophthalmology and Strabismus, 2005
To compare efficacy and complications of isolated unilateral superior oblique tucking in patients with unilateral superior oblique palsy (SOP).A retrospective analysis of 24 cases of unilateral SOP, 13 Acquired (group 1), and 11 Congenital (group 2), who underwent isolated unilateral superior oblique tuck over a 13-year period was performed.The mean ...
Rahul, Bhola, Federico G, Velez, Arthur L, Rosenbaum   +2 more
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Idiopathic Superior Oblique Palsies in Children

Journal of Pediatric Ophthalmology & Strabismus, 1990
ABSTRACT The records of 110 patients with superior oblique palsy seen at the Boston Children's Hospital between 1966 and 1988 were reviewed. Patients with identifiable orbital or neurological disorders affecting the fourth cranial nerve or with a history of head trauma preceding their superior oblique palsy were excluded from analysis ...
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Isolated superior oblique palsies with brainstem lesions

Neurology, 1999
Isolated unilateral superior oblique palsies resulting from brainstem lesions occurred in three patients. MRI documented contralateral tegmental lesions of the trochlear nucleus and adjacent intraaxial trochlear nerve. Lacunar infarct was the cause in two patients and a small hemorrhage in a third.
F, Thömke, K, Ringel
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