Results 121 to 130 of about 3,869,430 (327)

YAP1::TFE3 mediates endothelial‐to‐mesenchymal plasticity in epithelioid hemangioendothelioma

open access: yesMolecular Oncology, EarlyView.
The YAP1::TFE3 fusion protein drives endothelial‐to‐mesenchymal transition (EndMT) plasticity, resulting in the loss of endothelial characteristics and gain of mesenchymal‐like properties, including resistance to anoikis, increased migratory capacity, and loss of contact growth inhibition in endothelial cells.
Ant Murphy   +9 more
wiley   +1 more source

This School Works for Me: A Guide for Data Analysts [PDF]

open access: yes, 2010
Details the data collection, synthesis, and analyses required to identify at-risk students and gauge progress, assess school and program performance, and optimize resource allocation to raise graduation rates.

core  

Emerging role of ARHGAP29 in melanoma cell phenotype switching

open access: yesMolecular Oncology, EarlyView.
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster   +3 more
wiley   +1 more source

DİFFÉRANCE’IN SERÜVENİ

open access: yesFelsefe Dünyası, 2012
Bu çalışmanın temel amacı différance’ın kavram olmadığını, mevcudiyetmetafiziğine karşı indirgenemezliğini vurgulamaktır. Bu yüzden ne olduğununanlaşılması için ayrıntılı olarak ele alınmıştır.
Adem Yıldırım
doaj  

PYCR1 inhibition in bone marrow stromal cells enhances bortezomib sensitivity in multiple myeloma cells by altering their metabolism

open access: yesMolecular Oncology, EarlyView.
This study investigated how PYCR1 inhibition in bone marrow stromal cells (BMSCs) indirectly affects multiple myeloma (MM) cell metabolism and viability. Culturing MM cells in conditioned medium from PYCR1‐silenced BMSCs impaired oxidative phosphorylation and increased sensitivity to bortezomib.
Inge Oudaert   +13 more
wiley   +1 more source

Far infrared supplement: Catalog of infrared observations [PDF]

open access: yes
The Far Infrared Supplement: catalog of infrared observations summarizes all infrared astronomical observations at far infrared wavelengths published in the scientific literature between 1965 and 1982. The Supplement list contains 25% of the observations
Gezari, D. Y., Mead, J. M., Schmitz, M.
core   +1 more source

Carcaça e componentes corporais de cordeiros lactentes em pastejo de azevém suplementados com leguminosa ou concentrado

open access: yesArquivo Brasileiro de Medicina Veterinária e Zootecnia
RESUMO O objetivo desta pesquisa foi avaliar o efeito da suplementação privativa, com concentrado ou leguminosa, sobre as características da carcaça e dos componentes corporais de cordeiros lactentes mantidos em pastejo de azevém.
A.C.R.S. Pellegrin   +11 more
doaj   +1 more source

In vitro properties of patient serum predict clinical outcome after high dose rate brachytherapy of hepatocellular carcinoma

open access: yesMolecular Oncology, EarlyView.
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser   +14 more
wiley   +1 more source

The amelioration of the impact of physical fatigue on cognitive performance by phytochemicals : a thesis presented in partial fulfillment of the requirements for the degree of Master of Arts in Psychology at Massey University, Manawatu, New Zealand [PDF]

open access: yes, 2016
Fatigue is common in everyday life. It is experienced as either cognitive or physical fatigue, both of which are intertwined. Researchers are interested in investigating the ability of phytochemical supplementation to improve cognitive performance by ...
McKenzie, Olivia
core  

Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine

open access: yesMolecular Oncology, EarlyView.
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu   +18 more
wiley   +1 more source

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