Results 151 to 160 of about 2,733,914 (301)
INNOVATION AND SUSTAINABILITY IN PATIENT CARE IN THE ERA OF HEALTH RESOURCE MOBILIZATION. [PDF]
Artifon ELA +2 more
europepmc +1 more source
Natural products target the aging kidney in diabetic nephropathy by restoring the AMPK–SIRT1–Nrf2 axis, reducing oxidative stress, inflammation, fibrosis, and cellular senescence while enhancing mitochondrial biogenesis and antioxidant defenses.
Sherif Hamidu +8 more
wiley +1 more source
Identity and entitlement in accounts of (morally) normative and informational social influence for sustainability. [PDF]
Cooper L.
europepmc +1 more source
ABSTRACT As global populations age, cancer is increasingly becoming a leading cause of morbidity and mortality among older adults, particularly in low‐ and middle‐income countries (LMICs). Despite accounting for the majority of new cancer cases and deaths, older individuals remain underrepresented in cancer research, clinical guidelines, and health ...
Ibrahim Bidemi Abdullateef +2 more
wiley +1 more source
Challenging the Biomimetic Promise 2.0: Negative Spillover of Bio-Inspired Versus Sustainability Framing on Public Perceptions of Bio-Inspired Technologies. [PDF]
Fenn J +5 more
europepmc +1 more source
Aging Is a Key Driver for Adult Acute Myeloid Leukemia
Acute myeloid leukemia (AML) is a classical age‐related hematologic malignancy, and a key driver of AML is aging, which profoundly regulates intrinsic factors such as genomic instability, epigenetic reprogramming, and metabolic dysregulation, and alters bone marrow microenvironment.
Rong Yin, Haojian Zhang
wiley +1 more source
Quality as a catalyst to achieve environmentally sustainable healthcare. [PDF]
Ehsan S, Singh H.
europepmc +1 more source
Trends in Extreme Precipitation Events for the Northeastern United States 1948-2007 [PDF]
Spierre, Susan G., Wake, Cameron P.
core +2 more sources
Mutant NPM1 in Acute Myeloid Leukemia Initiation and Maintenance
NPM1 mutations drive acute myeloid leukemia by acting as neomorphic transcriptional regulators that cooperate with Menin–MLL and XPO1 to sustain HOX/MEIS1 expression and block differentiation. Targeting these mutant‐specific transcriptional dependencies provides a rational therapeutic strategy for NPM1‐mutated AML.
Yanan Jiang +3 more
wiley +1 more source

