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[Swine dysentery. 1. Occurrence of swine dysentery and isolation of the organisms].
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Evaluation of some dietary treatments designed to reduce the incidence of swine dysentery
Z. Durmic +5 more
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Veterinary Pathology, 2017
Swine dysentery is a severe enteric disease in pigs, which is characterized by bloody to mucoid diarrhea and associated with reduced growth performance and variable mortality. This disease is most often observed in grower-finisher pigs, wherein susceptible pigs develop a significant mucohemorrhagic typhlocolitis following infection with strongly ...
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Swine dysentery is a severe enteric disease in pigs, which is characterized by bloody to mucoid diarrhea and associated with reduced growth performance and variable mortality. This disease is most often observed in grower-finisher pigs, wherein susceptible pigs develop a significant mucohemorrhagic typhlocolitis following infection with strongly ...
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Lymphocyte stimulation in swine dysentery
Veterinary Immunology and Immunopathology, 1981In lymphocyte stimulation studies of pigs affected with swine dysentery (SD) all of the pigs gave significant response (P less than 0.05) to soluble antigen from Treponema hyodysenteriae. Swine infected with virulent or attenuated T. hyodysenteriae gave significant lymphocytic response 3 or 6 weeks after infection; uninfected pigs did not give a ...
E M, Jenkins, P L, Klesius
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Swine Dysentery: Pathogenicity of Treponema hyodysenteriae
American Journal of Veterinary Research, 1975SUMMARY When pure cultures of Treponema hyodysenteriae were orally inoculated into pigs, severe disease characteristic of swine dysentery developed. Less severe lesions were produced by oral inoculation of infective minced colon. Noninoculated pigs were used as controls.
R, Hughes, H J, Olander, C B, Williams
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Tierarztliche Praxis, 1984
The microbiological and serological properties of Treponema hyodysenteriae, its mediators of pathogenicity and the morphological changes in infected animals were presented. From these changes and from the disturbances of the intestinal function, the clinical symptoms could be derived.
K, Seeger, P, Klatt, N, Deutschländer
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The microbiological and serological properties of Treponema hyodysenteriae, its mediators of pathogenicity and the morphological changes in infected animals were presented. From these changes and from the disturbances of the intestinal function, the clinical symptoms could be derived.
K, Seeger, P, Klatt, N, Deutschländer
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Swine dysentery: more unknown than known
Veterinary Microbiology, 1995Swine dysentery (SD) is an economically important disease. It is caused by the spirochete Serpulina hyodysenteriae. In order to minimize the economic damage of SD, several methods to control this disease are recommended. Whereas hygienic measures and use of antimicrobials are actually practised for prevention, detection and exclusion of carriers of S ...
A A, ter Huurne, W, Gaastra
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Disease facts - Swine dysentery
Livestock, 2009Affected individuals are usually pyrexic, depressed and inappetant, with withdrawn flanks (Fig. 3), often with foul smelling bloody mucoid faeces running down the perineum. Untreated it can kill or alternatively leave a chronically affected individual that fails to thrive, ultimately requiring euthanasia.
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Passive protection of segmented swine colonic loops against swine dysentery
American Journal of Veterinary Research, 1985SUMMARY Swine-ligated loops were used to demonstrate passive protection against swine dysentery. Loops inoculated with immune sera containing complement and with homologous Treponema hyodysenteriae were normal at necropsy. Loops inoculated with heat-inactivated immune sera and heterologous T hyodysenteriae were not protected. Loops inoculated with heat-
L A, Joens +5 more
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Immunity to Swine Dysentery in Recovered Pigs
American Journal of Veterinary Research, 1979SUMMARY The immune status of 29 pigs recovered from swine dysentery (sd) was evaluated after reexposure to Treponema hyodysenteriae. Pigs which had recovered from sd and remained asymptomatic for 4 to 6, 9 to 13, and 16 to 17 weeks after initial inoculation were reexposed to 1.5 X 109 viable cells of T hyodysenteriae per pig.
L A, Joens, D L, Harris, D H, Baum
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